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’s thyroiditis: a stem cell hypothesis of papillary carcinoma oncogenesis. Human Pathology 35 465 –473. Bykov VJ , Issaeva N, Shilov A, Hultcrantz M, Pugacheva E, Chumakov P, Bergman J, Wiman KG & Selivanova G 2002 Restoration
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.0240 ). Nikiforova MN Kimura ET Gandhi M Biddinger PW Knauf JA Basolo F Zhu Z Giannini R Salvatore G Fusco A 2003 BRAF mutations in thyroid tumors are restricted to papillary carcinomas and anaplastic or poorly differentiated carcinomas arising from papillary
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.23.1.0455 ) 10.1210/edrv.23.1.0455 11844744 Biyi A Zaimi S Doudouh A 2016 Functioning metastases from thyroid papillary carcinoma in bone . Journal of Nuclear Medicine Technology 44 253 – 254 . ( https://doi.org/10.2967/jnmt.116
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Hormones and Cancer Group, Department of Endocrine Surgery, Department of Anatomical Pathology, Endocrinology, Department of Neuropathology, Clinical Cooperation Unit Neuropathology, Northern Cancer Translational Research Unit, University of Sydney, Cancer Genetics Laboratory, Kolling Institute of Medical Research and
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Hormones and Cancer Group, Department of Endocrine Surgery, Department of Anatomical Pathology, Endocrinology, Department of Neuropathology, Clinical Cooperation Unit Neuropathology, Northern Cancer Translational Research Unit, University of Sydney, Cancer Genetics Laboratory, Kolling Institute of Medical Research and
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Hormones and Cancer Group, Department of Endocrine Surgery, Department of Anatomical Pathology, Endocrinology, Department of Neuropathology, Clinical Cooperation Unit Neuropathology, Northern Cancer Translational Research Unit, University of Sydney, Cancer Genetics Laboratory, Kolling Institute of Medical Research and
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Hormones and Cancer Group, Department of Endocrine Surgery, Department of Anatomical Pathology, Endocrinology, Department of Neuropathology, Clinical Cooperation Unit Neuropathology, Northern Cancer Translational Research Unit, University of Sydney, Cancer Genetics Laboratory, Kolling Institute of Medical Research and
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Hormones and Cancer Group, Department of Endocrine Surgery, Department of Anatomical Pathology, Endocrinology, Department of Neuropathology, Clinical Cooperation Unit Neuropathology, Northern Cancer Translational Research Unit, University of Sydney, Cancer Genetics Laboratory, Kolling Institute of Medical Research and
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Hormones and Cancer Group, Department of Endocrine Surgery, Department of Anatomical Pathology, Endocrinology, Department of Neuropathology, Clinical Cooperation Unit Neuropathology, Northern Cancer Translational Research Unit, University of Sydney, Cancer Genetics Laboratory, Kolling Institute of Medical Research and
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Hormones and Cancer Group, Department of Endocrine Surgery, Department of Anatomical Pathology, Endocrinology, Department of Neuropathology, Clinical Cooperation Unit Neuropathology, Northern Cancer Translational Research Unit, University of Sydney, Cancer Genetics Laboratory, Kolling Institute of Medical Research and
Hormones and Cancer Group, Department of Endocrine Surgery, Department of Anatomical Pathology, Endocrinology, Department of Neuropathology, Clinical Cooperation Unit Neuropathology, Northern Cancer Translational Research Unit, University of Sydney, Cancer Genetics Laboratory, Kolling Institute of Medical Research and
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Hormones and Cancer Group, Department of Endocrine Surgery, Department of Anatomical Pathology, Endocrinology, Department of Neuropathology, Clinical Cooperation Unit Neuropathology, Northern Cancer Translational Research Unit, University of Sydney, Cancer Genetics Laboratory, Kolling Institute of Medical Research and
Hormones and Cancer Group, Department of Endocrine Surgery, Department of Anatomical Pathology, Endocrinology, Department of Neuropathology, Clinical Cooperation Unit Neuropathology, Northern Cancer Translational Research Unit, University of Sydney, Cancer Genetics Laboratory, Kolling Institute of Medical Research and
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Surgical Pathology 36 844 – 850 . ( doi:10.1097/PAS.0b013e318246b527 ). Lee JH Lee ES Kim YS 2007 Clinicopathologic significance of BRAF V600E mutation in papillary carcinomas of the thyroid: a meta-analysis . Cancer 110 38 – 46 . ( doi
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Manger et al . (1977) 15/F Acromegaly/PHEO – b NA Melicow (1977) 52/F Chromophobe adenoma of pituitary/papillary carcinoma of thyroid/PHEO diagnosed postmortem – b NA Janson et al . (1978) 28/F Pituitary adenoma (probably nonfunctioning)/bilateral PHEO
Institute of Pathology and Immunology of the University of Porto (IPATIMUP), Department of Pathology, Portugese Institute of Oncology, Department of Pathology, Rua Roberto Frias s/n, 4200-465 Porto, Portugal
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Institute of Pathology and Immunology of the University of Porto (IPATIMUP), Department of Pathology, Portugese Institute of Oncology, Department of Pathology, Rua Roberto Frias s/n, 4200-465 Porto, Portugal
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Institute of Pathology and Immunology of the University of Porto (IPATIMUP), Department of Pathology, Portugese Institute of Oncology, Department of Pathology, Rua Roberto Frias s/n, 4200-465 Porto, Portugal
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Institute of Pathology and Immunology of the University of Porto (IPATIMUP), Department of Pathology, Portugese Institute of Oncology, Department of Pathology, Rua Roberto Frias s/n, 4200-465 Porto, Portugal
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Institute of Pathology and Immunology of the University of Porto (IPATIMUP), Department of Pathology, Portugese Institute of Oncology, Department of Pathology, Rua Roberto Frias s/n, 4200-465 Porto, Portugal
Institute of Pathology and Immunology of the University of Porto (IPATIMUP), Department of Pathology, Portugese Institute of Oncology, Department of Pathology, Rua Roberto Frias s/n, 4200-465 Porto, Portugal
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cell lines . Oncogene 24 1455 – 1460 . doi:10.1038/sj.onc.1208292 . Albores-Saavedra J 2010 Papillary thyroid carcinoma with prominent hobnail features: a new aggressive variant of moderately differentiated papillary carcinoma. A
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). (A) Ranking of papillary carcinoma according to the BRS. (B) Driver mutational status. (C) Thyroid differentiation score. (D) Genomic platform clusters. (E) Histologic type and follicular fraction. Reproduced, with permission, from Cancer Genome
Endocrinology Service, Human Oncology and Pathogenesis Program, Department of Pathology, Department of Medicine
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Endocrinology Service, Human Oncology and Pathogenesis Program, Department of Pathology, Department of Medicine
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Endocrinology Service, Human Oncology and Pathogenesis Program, Department of Pathology, Department of Medicine
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-differentiated papillary carcinoma, follicular variant. (B) High power of (A) (arrow indicates isolated TAM). (C) Moderate density of CD68 + TAM in poorly differentiated thyroid carcinomas. (D) High power of (C) (arrow indicates TAM). (E) Very high density of CD68 + TAM
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), oral squamous cell carcinoma ( Piao et al . 2012 ), and papillary carcinoma ( Wang et al . 2013 ). The overexpression of USP22 is related to poor prognosis and may be used as a biomarker during diagnosis. USP22 has been identified as a cancer stem
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Porta G Fusco A Vecchio G 1990 PTC is a novel rearranged form of the ret proto-oncogene and is frequently detected in vivo in human thyroid papillary carcinomas . Cell 60 557 – 563 . ( https://doi.org/10.1016/0092-8674(90)90659-3 ) Haffter P