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E Patterson
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R Webb
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A Weisbrod
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B Bian
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M He
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L Zhang
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A K Holloway Endocrine Oncology Section, Gladstone Institutes, Program in Reproductive and Adult Endocrinology, NIH/NCI/Surgery Branch, National Cancer Institute, NIH, Hatfield Clinical Research Center, Room 4‐5952, 10 Center Drive, Bethesda, Maryland 20892, USA

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R Krishna
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N Nilubol
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K Pacak Endocrine Oncology Section, Gladstone Institutes, Program in Reproductive and Adult Endocrinology, NIH/NCI/Surgery Branch, National Cancer Institute, NIH, Hatfield Clinical Research Center, Room 4‐5952, 10 Center Drive, Bethesda, Maryland 20892, USA

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E Kebebew
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( Zarnegar et al . 2006 ). The prevalence of malignant pheochromocytoma is 2.5–40%, and the overall survival rate is <50% at 5 years ( Zarnegar et al . 2006 ). Currently, there are no reliable histologic or molecular markers for distinguishing between

Article Type:
Research Article
DOI:
https://doi.org/10.1530/ERC-11-0308
Volume/Issue:
Volume 19: Issue 2
Free access
Vincenzo Marotta Department of Clinical Medicine and Surgery, Federico II University, Naples, Italy

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Maria Chiara Zatelli Section of Endocrinology and Internal Medicine, Department of Medical Sciences, University of Ferrara, Ferrara, Italy

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Concetta Sciammarella IOS & COLEMAN Srl, Naples, Italy

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Maria Rosaria Ambrosio Section of Endocrinology and Internal Medicine, Department of Medical Sciences, University of Ferrara, Ferrara, Italy

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Marta Bondanelli Section of Endocrinology and Internal Medicine, Department of Medical Sciences, University of Ferrara, Ferrara, Italy

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Annamaria Colao Department of Clinical Medicine and Surgery, Federico II University, Naples, Italy

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Antongiulio Faggiano Thyroid and Parathyroid Surgery Unit, Istituto Nazionale per lo Studio e la Cura dei Tumori ‘Fondazione G. Pascale’ – IRCCS, Naples, Italy

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. Particularly, researchers aimed to identify markers useful for (a) diagnosis anticipation and refining, (b) prognostic stratification and (c) disease evolution monitoring and response to treatment. Based on the relationship with codified hormone

Article Type:
Review Article
DOI:
https://doi.org/10.1530/ERC-17-0269
Volume/Issue:
Volume 25: Issue 1
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Kwon Joong Na Department of Community Health, Korea Health Promotion Institute, Seoul, Republic of Korea
Department of Clinical Medical Sciences, Seoul National University, College of Medicine, Seoul, Republic of Korea

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Hongyoon Choi Department of Nuclear Medicine, Seoul National University Hospital, Seoul, Republic of Korea
Cheonan Public Health Center, Chungnam, Republic of Korea

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’s multiple testing corrections. Expression of immunosuppressive molecular markers, CTLA-4, PD-L1 and HLA-G, was statistically compared according to TDS and the BRAF V600E mutation status. The correlation between TDS and immunosuppressive markers was

Article Type:
Research Article
DOI:
https://doi.org/10.1530/ERC-17-0532
Volume/Issue:
Volume 25: Issue 5
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Chun-Peng Liao Lawrence J. Ellison Institute for Transformative Medicine, Keck School of Medicine, University of Southern California, Los Angeles, California, USA

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Leng-Ying Chen Lawrence J. Ellison Institute for Transformative Medicine, Keck School of Medicine, University of Southern California, Los Angeles, California, USA

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Andrea Luethy Lawrence J. Ellison Institute for Transformative Medicine, Keck School of Medicine, University of Southern California, Los Angeles, California, USA

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Youngsoo Kim Ionis Pharmaceuticals Inc., Carlsbad, California, USA

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Kian Kani Lawrence J. Ellison Institute for Transformative Medicine, Keck School of Medicine, University of Southern California, Los Angeles, California, USA

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A Robert MacLeod Ionis Pharmaceuticals Inc., Carlsbad, California, USA

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Mitchell E Gross Lawrence J. Ellison Institute for Transformative Medicine, Keck School of Medicine, University of Southern California, Los Angeles, California, USA

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attenuated by co-incubation with AR-depleted CAFs. Interestingly, the expression of several stem cell marker genes was upregulated in PCa cells grown with AR-depleted CAFs. Further, interferon gamma (IFN-γ) and macrophage colony-stimulating factor (M

Article Type:
Research Article
DOI:
https://doi.org/10.1530/ERC-16-0138
Volume/Issue:
Volume 24: Issue 4
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Maria Chiara Zatelli Section of Endocrinology and Internal Medicine, Neuroendocrine Tumours Unit, Endocrinology Unit, Department of Clinical Medicine and Surgery, Thyroid and Parathyroid Surgery Unit, Department of Medical Sciences, University of Ferrara, Via Aldo Moro 8, 44124 Cona – Ferrara, Italy

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Giuseppe Fanciulli Section of Endocrinology and Internal Medicine, Neuroendocrine Tumours Unit, Endocrinology Unit, Department of Clinical Medicine and Surgery, Thyroid and Parathyroid Surgery Unit, Department of Medical Sciences, University of Ferrara, Via Aldo Moro 8, 44124 Cona – Ferrara, Italy

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Pasqualino Malandrino Section of Endocrinology and Internal Medicine, Neuroendocrine Tumours Unit, Endocrinology Unit, Department of Clinical Medicine and Surgery, Thyroid and Parathyroid Surgery Unit, Department of Medical Sciences, University of Ferrara, Via Aldo Moro 8, 44124 Cona – Ferrara, Italy

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Valeria Ramundo Section of Endocrinology and Internal Medicine, Neuroendocrine Tumours Unit, Endocrinology Unit, Department of Clinical Medicine and Surgery, Thyroid and Parathyroid Surgery Unit, Department of Medical Sciences, University of Ferrara, Via Aldo Moro 8, 44124 Cona – Ferrara, Italy

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Antongiulio Faggiano Section of Endocrinology and Internal Medicine, Neuroendocrine Tumours Unit, Endocrinology Unit, Department of Clinical Medicine and Surgery, Thyroid and Parathyroid Surgery Unit, Department of Medical Sciences, University of Ferrara, Via Aldo Moro 8, 44124 Cona – Ferrara, Italy

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Annamaria Colao Section of Endocrinology and Internal Medicine, Neuroendocrine Tumours Unit, Endocrinology Unit, Department of Clinical Medicine and Surgery, Thyroid and Parathyroid Surgery Unit, Department of Medical Sciences, University of Ferrara, Via Aldo Moro 8, 44124 Cona – Ferrara, Italy

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on behalf of NIKE Group
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enteropancreatic neuroendocrine tumors . Anticancer Research 30 5063 – 5067 . Gilbert JA Adhikari LJ Lloyd RV Halfdanarson TR Muders MH Ames MM 2013 Molecular markers for novel therapeutic strategies in pancreatic endocrine tumors . Pancreas

Article Type:
Review Article
DOI:
https://doi.org/10.1530/ERC-15-0413
Volume/Issue:
Volume 23: Issue 3
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Mojun Zhu Department of Oncology, Mayo Clinic, Rochester, Minnesota, USA

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Karl R Sorenson Department of Oncology, Mayo Clinic, Rochester, Minnesota, USA

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Rebecca Liu Department of Medicine, Massachusetts General Hospital, Boston, Massachusetts, USA

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Bonnie E Gould Rothberg Smilow Cancer Hospital, Yale-New Haven Health System, New Haven, Connecticut, USA

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Thorvardur R Halfdanarson Department of Oncology, Mayo Clinic, Rochester, Minnesota, USA

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Search strategy We searched the PubMed database through February 1, 2021, in order to identify primary research articles that analyzed molecular markers as PNET prognostic factors. A non-redundant list of candidate articles was created from the union of

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https://doi.org/10.1530/ERC-21-0075
Volume/Issue:
Volume 28: Issue 12
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Ta-Chun Yuan
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Suresh Veeramani
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Fen-Fen Lin
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Dmitry Kondrikou
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Stanislav Zelivianski
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Tsukasa Igawa
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Dev Karan
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Surinder K. Batra
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Ming-Fong Lin
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cytoplasm and secrete several neuronal markers as well as growth-stimulatory factors, while they lack the expression of androgen receptor (AR). It is proposed that these NE cells originate from endodermal epithelial cells and are terminally differentiated

Article Type:
Research Article
DOI:
https://doi.org/10.1677/erc.1.01043
Volume/Issue:
Volume 13: Issue 1
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Hen Prizant Division of Endocrinology and Metabolism, Department of Medicine, University of Rochester Medical Center, Rochester, New York, USA

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Manisha Taya Division of Endocrinology and Metabolism, Department of Medicine, University of Rochester Medical Center, Rochester, New York, USA

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Irina Lerman Division of Endocrinology and Metabolism, Department of Medicine, University of Rochester Medical Center, Rochester, New York, USA

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Allison Light Division of Endocrinology and Metabolism, Department of Medicine, University of Rochester Medical Center, Rochester, New York, USA

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Aritro Sen Division of Endocrinology and Metabolism, Department of Medicine, University of Rochester Medical Center, Rochester, New York, USA

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Soumya Mitra Department of Imaging Sciences, University of Rochester Medical Center, Rochester, New York, USA

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Thomas H Foster Department of Imaging Sciences, University of Rochester Medical Center, Rochester, New York, USA

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Stephen R Hammes Division of Endocrinology and Metabolism, Department of Medicine, University of Rochester Medical Center, Rochester, New York, USA

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TSC2 mutations, LAM cells typically express melanocytic markers. One such marker, gp100 (PMEL), is often up-regulated in LAM cells ( Kuhnen et al . 2001 ). PMEL is the target for the HMB-45 antibody; thus, HMB-45 staining by immunohistochemistry (IHC

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Research Article
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https://doi.org/10.1530/ERC-15-0505
Volume/Issue:
Volume 23: Issue 4
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Jennifer W Carlisle Winship Cancer Institute of Emory University, Atlanta, Georgia, USA
Department of Hematology and Oncology, Emory University School of Medicine, Atlanta, Georgia, USA

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Caroline S Jansen Winship Cancer Institute of Emory University, Atlanta, Georgia, USA
Department of Urology, Emory University School of Medicine, Atlanta, Georgia, USA
Emory Vaccine Center, Emory University School of Medicine, Atlanta, Georgia, USA

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Mehmet Asim Bilen Winship Cancer Institute of Emory University, Atlanta, Georgia, USA
Department of Hematology and Oncology, Emory University School of Medicine, Atlanta, Georgia, USA

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Haydn Kissick Winship Cancer Institute of Emory University, Atlanta, Georgia, USA
Department of Urology, Emory University School of Medicine, Atlanta, Georgia, USA
Department of Microbiology and Immunology, Emory University School of Medicine, Atlanta, Georgia, USA
Emory Vaccine Center, Emory University School of Medicine, Atlanta, Georgia, USA

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considered. Herein, we review the T-cell response to acute viral infection, explore the overlap among markers of T-cell activation in response to immunotherapy and in viral infections, highlight similarities in systemic inflammatory profiles in these two

Article Type:
Article Commentary
DOI:
https://doi.org/10.1530/ERC-20-0187
Volume/Issue:
Volume 27: Issue 9
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Y M H Jonkers
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S M H Claessen
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A Perren
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A M Schmitt
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L J Hofland
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W de Herder
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R R de Krijger
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A A J Verhofstad
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A R Hermus
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J A Kummer
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B Skogseid
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M Volante
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A C Voogd
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F C S Ramaekers
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E J M Speel
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is also expressed in benign tumors, this marker is considered to be of limited value ( Heitz et al. 1987 , Graeme-Cook et al. 1990 ). Up-regulation of COX2 and CK19, and down-regulation of p27 and CD99 were found to be associated with Ki67

Article Type:
Research Article
DOI:
https://doi.org/10.1677/ERC-07-0111
Volume/Issue:
Volume 14: Issue 3
Free access