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Mark A White Center for Nuclear Receptors and Cell Signaling, University of Houston, Houston, Texas, USA
Department of Biology and Biochemistry, University of Houston, Houston, Texas, USA

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Efrosini Tsouko Center for Nuclear Receptors and Cell Signaling, University of Houston, Houston, Texas, USA
Department of Biology and Biochemistry, University of Houston, Houston, Texas, USA

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Chenchu Lin Department of Cancer Systems Imaging, The University of Texas M.D. Anderson Cancer Center, Houston, Texas, USA
The University of Texas MD Anderson Cancer Center UTHealth Graduate School of Biomedical Sciences, Houston, Texas, USA

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Kimal Rajapakshe Department of Molecular and Cellular Biology, Baylor College of Medicine, Houston, Texas, USA

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Jeffrey M Spencer Center for Nuclear Receptors and Cell Signaling, University of Houston, Houston, Texas, USA
Department of Biology and Biochemistry, University of Houston, Houston, Texas, USA

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Sandi R Wilkenfeld Department of Cancer Systems Imaging, The University of Texas M.D. Anderson Cancer Center, Houston, Texas, USA
The University of Texas MD Anderson Cancer Center UTHealth Graduate School of Biomedical Sciences, Houston, Texas, USA

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Sheiva S Vakili Center for Nuclear Receptors and Cell Signaling, University of Houston, Houston, Texas, USA
Department of Biology and Biochemistry, University of Houston, Houston, Texas, USA

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Thomas L Pulliam Center for Nuclear Receptors and Cell Signaling, University of Houston, Houston, Texas, USA
Department of Biology and Biochemistry, University of Houston, Houston, Texas, USA
Department of Cancer Systems Imaging, The University of Texas M.D. Anderson Cancer Center, Houston, Texas, USA

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Dominik Awad Department of Cancer Systems Imaging, The University of Texas M.D. Anderson Cancer Center, Houston, Texas, USA
The University of Texas MD Anderson Cancer Center UTHealth Graduate School of Biomedical Sciences, Houston, Texas, USA

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Fotis Nikolos Department of Molecular and Cellular Biology, Baylor College of Medicine, Houston, Texas, USA

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Rajasekhara Reddy Katreddy Department of Biology and Biochemistry, University of Houston, Houston, Texas, USA

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Benny Abraham Kaipparettu Department of Molecular and Human Genetics, Baylor College of Medicine, Houston, Texas, USA
Dan L. Duncan Cancer Center, Baylor College of Medicine, Houston, Texas, USA

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Arun Sreekumar Department of Molecular and Cellular Biology, Baylor College of Medicine, Houston, Texas, USA
Dan L. Duncan Cancer Center, Baylor College of Medicine, Houston, Texas, USA

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Xiaoliu Zhang Center for Nuclear Receptors and Cell Signaling, University of Houston, Houston, Texas, USA
Department of Biology and Biochemistry, University of Houston, Houston, Texas, USA

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Edwin Cheung Biology and Pharmacology, Genome Institute of Singapore, A*STAR, Singapore, Singapore
Faculty of Health Sciences, University of Macau, Taipa, Macau, China

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Cristian Coarfa Department of Molecular and Cellular Biology, Baylor College of Medicine, Houston, Texas, USA

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Daniel E Frigo Center for Nuclear Receptors and Cell Signaling, University of Houston, Houston, Texas, USA
Department of Biology and Biochemistry, University of Houston, Houston, Texas, USA
Department of Cancer Systems Imaging, The University of Texas M.D. Anderson Cancer Center, Houston, Texas, USA
Department of Genitourinary Medical Oncology, The University of Texas M.D. Anderson Cancer Center, Houston, Texas, USA
Molecular Medicine Program, The Houston Methodist Research Institute, Houston, Texas, USA

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( Kramer et al. 2006 , Treebak et al. 2006 ), to our knowledge, the androgen and CaMKK2-AMPK modulation of TBC1D4 expression has not been previously described. Ultimately, this combined series of transcriptional and posttranscriptional events converge

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Belinda J Petri Department of Biochemistry & Molecular Genetics, University of Louisville School of Medicine Louisville, Kentucky, USA

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Kellianne M Piell Department of Biochemistry & Molecular Genetics, University of Louisville School of Medicine Louisville, Kentucky, USA

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Ali E Wilt Department of Biochemistry & Molecular Genetics, University of Louisville School of Medicine Louisville, Kentucky, USA

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Alexa D Howser Department of Biochemistry & Molecular Genetics, University of Louisville School of Medicine Louisville, Kentucky, USA

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Laura Winkler Department of Biochemistry & Molecular Genetics, University of Louisville School of Medicine Louisville, Kentucky, USA

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Mattie R Whitworth Department of Biochemistry & Molecular Genetics, University of Louisville School of Medicine Louisville, Kentucky, USA

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Bailey L Valdes Department of Biochemistry & Molecular Genetics, University of Louisville School of Medicine Louisville, Kentucky, USA

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Norman L Lehman Department of Biochemistry & Molecular Genetics, University of Louisville School of Medicine Louisville, Kentucky, USA
Pathology and Laboratory Medicine, University of Louisville, Louisville, Kentucky, USA
The Brown Cancer Center, University of Louisville, Louisville, Kentucky, USA

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Brian F Clem Department of Biochemistry & Molecular Genetics, University of Louisville School of Medicine Louisville, Kentucky, USA
The Brown Cancer Center, University of Louisville, Louisville, Kentucky, USA

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Carolyn M Klinge Department of Biochemistry & Molecular Genetics, University of Louisville School of Medicine Louisville, Kentucky, USA
The Brown Cancer Center, University of Louisville, Louisville, Kentucky, USA
Center for Integrative Environmental Health Sciences (CIEHS), University of Louisville, Louisville, Kentucky, USA

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and mRNA transcript abundance in these BCa cell lines suggest posttranscriptional modulation of PHGDH protein expression. A2B1 expression decreases miRNAs predicted to target PSAT1 and PHGDH Gene ontology identified ‘serine family amino acid

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S C J P Gielen
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L C M Kühne
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P C Ewing
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L J Blok
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C W Burger
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-related protein complex 1, sigma 2 subunit AA205444 Hs.121592 0.44094 0.022774 APG7L APG7 autophagy 7-like ( S. cerevisiae

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Luiz Gustavo A Chuffa Department of Anatomy, Department of Morphology and Pathology, Department of Biological Sciences, Institute of Biosciences of Botucatu, UNESP – Universidade Estadual Paulista, PO Box 18618-970, Rubião Júnior, s/n, Botucatu, São Paulo 510, Brazil

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Michelly S Alves Department of Anatomy, Department of Morphology and Pathology, Department of Biological Sciences, Institute of Biosciences of Botucatu, UNESP – Universidade Estadual Paulista, PO Box 18618-970, Rubião Júnior, s/n, Botucatu, São Paulo 510, Brazil

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Marcelo Martinez Department of Anatomy, Department of Morphology and Pathology, Department of Biological Sciences, Institute of Biosciences of Botucatu, UNESP – Universidade Estadual Paulista, PO Box 18618-970, Rubião Júnior, s/n, Botucatu, São Paulo 510, Brazil

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Isabel Cristina C Camargo Department of Anatomy, Department of Morphology and Pathology, Department of Biological Sciences, Institute of Biosciences of Botucatu, UNESP – Universidade Estadual Paulista, PO Box 18618-970, Rubião Júnior, s/n, Botucatu, São Paulo 510, Brazil

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Patricia F F Pinheiro Department of Anatomy, Department of Morphology and Pathology, Department of Biological Sciences, Institute of Biosciences of Botucatu, UNESP – Universidade Estadual Paulista, PO Box 18618-970, Rubião Júnior, s/n, Botucatu, São Paulo 510, Brazil

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Raquel F Domeniconi Department of Anatomy, Department of Morphology and Pathology, Department of Biological Sciences, Institute of Biosciences of Botucatu, UNESP – Universidade Estadual Paulista, PO Box 18618-970, Rubião Júnior, s/n, Botucatu, São Paulo 510, Brazil

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Luiz Antonio L Júnior Department of Anatomy, Department of Morphology and Pathology, Department of Biological Sciences, Institute of Biosciences of Botucatu, UNESP – Universidade Estadual Paulista, PO Box 18618-970, Rubião Júnior, s/n, Botucatu, São Paulo 510, Brazil

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Francisco Eduardo Martinez Department of Anatomy, Department of Morphology and Pathology, Department of Biological Sciences, Institute of Biosciences of Botucatu, UNESP – Universidade Estadual Paulista, PO Box 18618-970, Rubião Júnior, s/n, Botucatu, São Paulo 510, Brazil

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et al . 2002 , Batista et al . 2013 , Fan et al . 2013 , Hong et al . 2014 ). Activities of Mel on such tumors include modulation of BAX and Bcl-2 ( Hong et al . 2014 ), p53 and p21 ( Cos et al . 2002 ), and further regulation of caspase-3

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Kristen Wong Department of Developmental and Molecular Biology, Albert Einstein College of Medicine, Bronx, New York, USA

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Francesca Di Cristofano Department of Developmental and Molecular Biology, Albert Einstein College of Medicine, Bronx, New York, USA

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Michela Ranieri Department of Developmental and Molecular Biology, Albert Einstein College of Medicine, Bronx, New York, USA

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Daniela De Martino Department of Developmental and Molecular Biology, Albert Einstein College of Medicine, Bronx, New York, USA

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Antonio Di Cristofano Department of Developmental and Molecular Biology, Albert Einstein College of Medicine, Bronx, New York, USA

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.1158/1535-7163.MCT-05-0363 16985050 Guarducci C Bonechi M Benelli M Biagioni C Boccalini G Romagnoli D Verardo R Schiff R Osborne CK De Angelis C , et al . 2018 Cyclin E1 and Rb modulation as common events at time of resistance to palbociclib in hormone

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Yan Huang Department of Gynecology, Department of Gynecologic Oncology, Systems Biology, Experimental Therapeutics, Department of Obstetrics and Gynecology, Obstetrics and Gynecology Hospital of Fudan University, 419 Fang Xie Road, Shanghai 200011, People's Republic of China
Department of Gynecology, Department of Gynecologic Oncology, Systems Biology, Experimental Therapeutics, Department of Obstetrics and Gynecology, Obstetrics and Gynecology Hospital of Fudan University, 419 Fang Xie Road, Shanghai 200011, People's Republic of China

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Hongyan Jin Department of Gynecology, Department of Gynecologic Oncology, Systems Biology, Experimental Therapeutics, Department of Obstetrics and Gynecology, Obstetrics and Gynecology Hospital of Fudan University, 419 Fang Xie Road, Shanghai 200011, People's Republic of China

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Yingtao Liu Department of Gynecology, Department of Gynecologic Oncology, Systems Biology, Experimental Therapeutics, Department of Obstetrics and Gynecology, Obstetrics and Gynecology Hospital of Fudan University, 419 Fang Xie Road, Shanghai 200011, People's Republic of China

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Jiayi Zhou Department of Gynecology, Department of Gynecologic Oncology, Systems Biology, Experimental Therapeutics, Department of Obstetrics and Gynecology, Obstetrics and Gynecology Hospital of Fudan University, 419 Fang Xie Road, Shanghai 200011, People's Republic of China

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Jingxin Ding Department of Gynecology, Department of Gynecologic Oncology, Systems Biology, Experimental Therapeutics, Department of Obstetrics and Gynecology, Obstetrics and Gynecology Hospital of Fudan University, 419 Fang Xie Road, Shanghai 200011, People's Republic of China

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Kwai Wa Cheng Department of Gynecology, Department of Gynecologic Oncology, Systems Biology, Experimental Therapeutics, Department of Obstetrics and Gynecology, Obstetrics and Gynecology Hospital of Fudan University, 419 Fang Xie Road, Shanghai 200011, People's Republic of China

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Yinhua Yu Department of Gynecology, Department of Gynecologic Oncology, Systems Biology, Experimental Therapeutics, Department of Obstetrics and Gynecology, Obstetrics and Gynecology Hospital of Fudan University, 419 Fang Xie Road, Shanghai 200011, People's Republic of China
Department of Gynecology, Department of Gynecologic Oncology, Systems Biology, Experimental Therapeutics, Department of Obstetrics and Gynecology, Obstetrics and Gynecology Hospital of Fudan University, 419 Fang Xie Road, Shanghai 200011, People's Republic of China

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Youji Feng Department of Gynecology, Department of Gynecologic Oncology, Systems Biology, Experimental Therapeutics, Department of Obstetrics and Gynecology, Obstetrics and Gynecology Hospital of Fudan University, 419 Fang Xie Road, Shanghai 200011, People's Republic of China
Department of Gynecology, Department of Gynecologic Oncology, Systems Biology, Experimental Therapeutics, Department of Obstetrics and Gynecology, Obstetrics and Gynecology Hospital of Fudan University, 419 Fang Xie Road, Shanghai 200011, People's Republic of China

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suggested a model of FSH–VEGF stimulation in ovarian cancer ( Huang et al . 2008 ). With the results obtained in the current study, we can supplement this model as shown in Fig. 8 . Our ongoing studies indicate that FSH stimulation also inhibits autophagy

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Jing Jing Department of Medical Oncology, Harbin Medical University Cancer Hospital, Harbin Medical University, Harbin, China

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Dandan Zhu Department of Medical Oncology, Harbin Medical University Cancer Hospital, Harbin Medical University, Harbin, China

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Xiaowei Wang Department of Medical Oncology, Harbin Medical University Cancer Hospital, Harbin Medical University, Harbin, China

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Jing An Institute of Cancer Prevention and Treatment, Heilongjiang Province Academy of Medical Sciences, Harbin, China
Oncology Key Lab of the Heilongjiang Province Institution of Higher Education, Harbin, China
Institute of Cancer Prevention and Treatment, Harbin Medical University Cancer Hospital, Harbin Medical University, Harbin, China

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Zhaoliang Liu Institute of Cancer Prevention and Treatment, Heilongjiang Province Academy of Medical Sciences, Harbin, China
Oncology Key Lab of the Heilongjiang Province Institution of Higher Education, Harbin, China
Institute of Cancer Prevention and Treatment, Harbin Medical University Cancer Hospital, Harbin Medical University, Harbin, China

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Qingyuan Zhang Department of Medical Oncology, Harbin Medical University Cancer Hospital, Harbin Medical University, Harbin, China
Institute of Cancer Prevention and Treatment, Heilongjiang Province Academy of Medical Sciences, Harbin, China
Oncology Key Lab of the Heilongjiang Province Institution of Higher Education, Harbin, China
Institute of Cancer Prevention and Treatment, Harbin Medical University Cancer Hospital, Harbin Medical University, Harbin, China

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tumor B cell infiltration might be a critical factor for HTBC patients, suggesting that modulation of B cell activity might be a potential way to address HT resistance in BC. An interesting discovery of our study is the association of the AS and SF

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Lei Qiao Department of Breast and Thyroid Surgery, Xinjiang Medical University Affiliated Tumor Hospital, Urumqi, Xinjiang Uygur Autonomous Region, China

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Chao Dong Department of Breast and Thyroid Surgery, Xinjiang Medical University Affiliated Tumor Hospital, Urumqi, Xinjiang Uygur Autonomous Region, China

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Wenlei Jia Department of Breast and Thyroid Surgery, Xinjiang Medical University Affiliated Tumor Hospital, Urumqi, Xinjiang Uygur Autonomous Region, China

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Gang Sun Department of Breast and Thyroid Surgery, Xinjiang Medical University Affiliated Tumor Hospital, Urumqi, Xinjiang Uygur Autonomous Region, China

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, including inducing cellular senescence or autophagy, altering drug uptake or efflux, activating cancer stem cells, strengthening DNA damage repair, inactivating apoptosis, etc. ( O’Reilly et al. 2015 , Nies et al. 2015 , Nath et al. 2022

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Ales Vicha
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David Taieb Department of Pediatric Hematology and Oncology, Service Central de Biophysique et de Médecine Nucléaire, Département d'Oncologie Moléculaire, Program in Reproductive and Adult Endocrinology, 2nd Medical School, Charles University and University Hospital Motol, Prague, Czech Republic
Department of Pediatric Hematology and Oncology, Service Central de Biophysique et de Médecine Nucléaire, Département d'Oncologie Moléculaire, Program in Reproductive and Adult Endocrinology, 2nd Medical School, Charles University and University Hospital Motol, Prague, Czech Republic

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Karel Pacak Department of Pediatric Hematology and Oncology, Service Central de Biophysique et de Médecine Nucléaire, Département d'Oncologie Moléculaire, Program in Reproductive and Adult Endocrinology, 2nd Medical School, Charles University and University Hospital Motol, Prague, Czech Republic

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expression as a critical tumorigenic mechanism. These modulations in the pseudohypoxic signature observed in SDHx -related tumors can distinguish the gene expression phenotypes observed in the two subgroups of tumors in cluster 1. Reactive oxygen species A

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S Kato Division of Obstetrics and Gynecology, Faculty of Medicine, Pontificia Universidad Católica de Chile (PUC), Santiago, Chile

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M F Liberona Division of Obstetrics and Gynecology, Faculty of Medicine, Pontificia Universidad Católica de Chile (PUC), Santiago, Chile

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J Cerda-Infante Department of Hematology and Oncology, Faculty of Medicine, PUC, Santiago, Chile
Department of Cellular and Molecular, Faculty of Biological Sciences, PUC, Santiago, Chile

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M Sánchez Department of Hematology and Oncology, Faculty of Medicine, PUC, Santiago, Chile

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J Henríquez Department of Hematology and Oncology, Faculty of Medicine, PUC, Santiago, Chile

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C Bizama Department of Pathology, Faculty of Medicine, PUC, Santiago, Chile

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M L Bravo Department of Physiological Sciences, Faculty of Biological Sciences, PUC, Santiago, Chile
Millennium Institute on Immunology and Immunotherapy, PUC, Santiago, Chile

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P Gonzalez Department of Physiological Sciences, Faculty of Biological Sciences, PUC, Santiago, Chile

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R Gejman Department of Pathology, Faculty of Medicine, PUC, Santiago, Chile

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J Brañes Division of Obstetrics and Gynecology, Faculty of Medicine, Pontificia Universidad Católica de Chile (PUC), Santiago, Chile

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K García Division of Obstetrics and Gynecology, Faculty of Medicine, Pontificia Universidad Católica de Chile (PUC), Santiago, Chile

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C Ibañez Department of Hematology and Oncology, Faculty of Medicine, PUC, Santiago, Chile
Millennium Institute on Immunology and Immunotherapy, PUC, Santiago, Chile

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G I Owen Department of Physiological Sciences, Faculty of Biological Sciences, PUC, Santiago, Chile
Millennium Institute on Immunology and Immunotherapy, PUC, Santiago, Chile

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J C Roa Department of Pathology, Faculty of Medicine, PUC, Santiago, Chile
Millennium Institute on Immunology and Immunotherapy, PUC, Santiago, Chile

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V Montecinos Department of Hematology and Oncology, Faculty of Medicine, PUC, Santiago, Chile

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M A Cuello Division of Obstetrics and Gynecology, Faculty of Medicine, Pontificia Universidad Católica de Chile (PUC), Santiago, Chile

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mevalonate and cholesterol synthesis and blocking protein prenylation by downstream depletion of FPP and GGP ( Berndt et al . 2011 ). Treating mut-TP53 HGS-ovC cell lines with increasing concentrations of statins induces autophagy, cell death and drug

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