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Department of Biology and Biochemistry, University of Houston, Houston, Texas, USA
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Department of Biology and Biochemistry, University of Houston, Houston, Texas, USA
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The University of Texas MD Anderson Cancer Center UTHealth Graduate School of Biomedical Sciences, Houston, Texas, USA
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Department of Biology and Biochemistry, University of Houston, Houston, Texas, USA
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The University of Texas MD Anderson Cancer Center UTHealth Graduate School of Biomedical Sciences, Houston, Texas, USA
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Department of Biology and Biochemistry, University of Houston, Houston, Texas, USA
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Department of Biology and Biochemistry, University of Houston, Houston, Texas, USA
Department of Cancer Systems Imaging, The University of Texas M.D. Anderson Cancer Center, Houston, Texas, USA
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The University of Texas MD Anderson Cancer Center UTHealth Graduate School of Biomedical Sciences, Houston, Texas, USA
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Dan L. Duncan Cancer Center, Baylor College of Medicine, Houston, Texas, USA
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Dan L. Duncan Cancer Center, Baylor College of Medicine, Houston, Texas, USA
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Department of Biology and Biochemistry, University of Houston, Houston, Texas, USA
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Faculty of Health Sciences, University of Macau, Taipa, Macau, China
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Department of Biology and Biochemistry, University of Houston, Houston, Texas, USA
Department of Cancer Systems Imaging, The University of Texas M.D. Anderson Cancer Center, Houston, Texas, USA
Department of Genitourinary Medical Oncology, The University of Texas M.D. Anderson Cancer Center, Houston, Texas, USA
Molecular Medicine Program, The Houston Methodist Research Institute, Houston, Texas, USA
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( Kramer et al. 2006 , Treebak et al. 2006 ), to our knowledge, the androgen and CaMKK2-AMPK modulation of TBC1D4 expression has not been previously described. Ultimately, this combined series of transcriptional and posttranscriptional events converge
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Pathology and Laboratory Medicine, University of Louisville, Louisville, Kentucky, USA
The Brown Cancer Center, University of Louisville, Louisville, Kentucky, USA
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The Brown Cancer Center, University of Louisville, Louisville, Kentucky, USA
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The Brown Cancer Center, University of Louisville, Louisville, Kentucky, USA
Center for Integrative Environmental Health Sciences (CIEHS), University of Louisville, Louisville, Kentucky, USA
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and mRNA transcript abundance in these BCa cell lines suggest posttranscriptional modulation of PHGDH protein expression. A2B1 expression decreases miRNAs predicted to target PSAT1 and PHGDH Gene ontology identified ‘serine family amino acid
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-related protein complex 1, sigma 2 subunit AA205444 Hs.121592 0.44094 0.022774 APG7L APG7 autophagy 7-like ( S. cerevisiae
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et al . 2002 , Batista et al . 2013 , Fan et al . 2013 , Hong et al . 2014 ). Activities of Mel on such tumors include modulation of BAX and Bcl-2 ( Hong et al . 2014 ), p53 and p21 ( Cos et al . 2002 ), and further regulation of caspase-3
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.1158/1535-7163.MCT-05-0363 16985050 Guarducci C Bonechi M Benelli M Biagioni C Boccalini G Romagnoli D Verardo R Schiff R Osborne CK De Angelis C , et al . 2018 Cyclin E1 and Rb modulation as common events at time of resistance to palbociclib in hormone
Department of Gynecology, Department of Gynecologic Oncology, Systems Biology, Experimental Therapeutics, Department of Obstetrics and Gynecology, Obstetrics and Gynecology Hospital of Fudan University, 419 Fang Xie Road, Shanghai 200011, People's Republic of China
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Department of Gynecology, Department of Gynecologic Oncology, Systems Biology, Experimental Therapeutics, Department of Obstetrics and Gynecology, Obstetrics and Gynecology Hospital of Fudan University, 419 Fang Xie Road, Shanghai 200011, People's Republic of China
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Department of Gynecology, Department of Gynecologic Oncology, Systems Biology, Experimental Therapeutics, Department of Obstetrics and Gynecology, Obstetrics and Gynecology Hospital of Fudan University, 419 Fang Xie Road, Shanghai 200011, People's Republic of China
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suggested a model of FSH–VEGF stimulation in ovarian cancer ( Huang et al . 2008 ). With the results obtained in the current study, we can supplement this model as shown in Fig. 8 . Our ongoing studies indicate that FSH stimulation also inhibits autophagy
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Oncology Key Lab of the Heilongjiang Province Institution of Higher Education, Harbin, China
Institute of Cancer Prevention and Treatment, Harbin Medical University Cancer Hospital, Harbin Medical University, Harbin, China
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Oncology Key Lab of the Heilongjiang Province Institution of Higher Education, Harbin, China
Institute of Cancer Prevention and Treatment, Harbin Medical University Cancer Hospital, Harbin Medical University, Harbin, China
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Institute of Cancer Prevention and Treatment, Heilongjiang Province Academy of Medical Sciences, Harbin, China
Oncology Key Lab of the Heilongjiang Province Institution of Higher Education, Harbin, China
Institute of Cancer Prevention and Treatment, Harbin Medical University Cancer Hospital, Harbin Medical University, Harbin, China
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tumor B cell infiltration might be a critical factor for HTBC patients, suggesting that modulation of B cell activity might be a potential way to address HT resistance in BC. An interesting discovery of our study is the association of the AS and SF
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, including inducing cellular senescence or autophagy, altering drug uptake or efflux, activating cancer stem cells, strengthening DNA damage repair, inactivating apoptosis, etc. ( O’Reilly et al. 2015 , Nies et al. 2015 , Nath et al. 2022
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Department of Pediatric Hematology and Oncology, Service Central de Biophysique et de Médecine Nucléaire, Département d'Oncologie Moléculaire, Program in Reproductive and Adult Endocrinology, 2nd Medical School, Charles University and University Hospital Motol, Prague, Czech Republic
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expression as a critical tumorigenic mechanism. These modulations in the pseudohypoxic signature observed in SDHx -related tumors can distinguish the gene expression phenotypes observed in the two subgroups of tumors in cluster 1. Reactive oxygen species A
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Department of Cellular and Molecular, Faculty of Biological Sciences, PUC, Santiago, Chile
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Millennium Institute on Immunology and Immunotherapy, PUC, Santiago, Chile
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Millennium Institute on Immunology and Immunotherapy, PUC, Santiago, Chile
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Millennium Institute on Immunology and Immunotherapy, PUC, Santiago, Chile
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Millennium Institute on Immunology and Immunotherapy, PUC, Santiago, Chile
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mevalonate and cholesterol synthesis and blocking protein prenylation by downstream depletion of FPP and GGP ( Berndt et al . 2011 ). Treating mut-TP53 HGS-ovC cell lines with increasing concentrations of statins induces autophagy, cell death and drug