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Laboratory of Immunopharmacology, Department of Basic Sciences, School of Dentistry, São Paulo State University (Unesp), Araçatuba, São Paulo, Brazil
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Laboratory of Immunopharmacology, Department of Basic Sciences, School of Dentistry, São Paulo State University (Unesp), Araçatuba, São Paulo, Brazil
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Bhutia SK 2018 Autophagy regulates cisplatin-induced stemness and chemoresistance via the upregulation of CD44, ABCB1 and ADAM17 in oral squamous cell carcinoma . Cell Proliferation 51 e12411. ( https://doi.org/10.1111/cpr.12411 ) Nilsson MB Sun
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Chemistry 281 19762 – 19771 . ( https://doi.org/10.1074/jbc.M600746200 ) Singh P Godbole M Rao G Annarao S Mitra K Roy R Ingle A Agarwal G Tiwari S 2011 Inhibition of autophagy stimulate molecular iodine-induced apoptosis in hormone
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thyroid carcinoma. Treatment outcome and prognostic factors . Cancer 103 1330 – 1335 . doi:10.1002/cncr.20936 . Lista P Straface E Brunelleschi S Franconi F Malorni W 2011 On the role of autophagy in human diseases: a gender perspective
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noted in bold. Discussion Survivin, an apoptosis inhibitor protein, is highly expressed in human cancers, including breast cancer, and is considered as a new therapeutic target. Recently, a critical role for Survivin in the control of autophagy was also
Children's Hospital, University of Helsinki, Program for Women's Health, Department of Obstetrics and Gynecology, Department of Pathology, Institute of Biomedicine and Research Program in Genome-Scale Biology, Department of Pediatrics, PO Box 22 (Stenbäckinkatu 11), 00014 Helsinki, Finland
Children's Hospital, University of Helsinki, Program for Women's Health, Department of Obstetrics and Gynecology, Department of Pathology, Institute of Biomedicine and Research Program in Genome-Scale Biology, Department of Pediatrics, PO Box 22 (Stenbäckinkatu 11), 00014 Helsinki, Finland
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Children's Hospital, University of Helsinki, Program for Women's Health, Department of Obstetrics and Gynecology, Department of Pathology, Institute of Biomedicine and Research Program in Genome-Scale Biology, Department of Pediatrics, PO Box 22 (Stenbäckinkatu 11), 00014 Helsinki, Finland
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Children's Hospital, University of Helsinki, Program for Women's Health, Department of Obstetrics and Gynecology, Department of Pathology, Institute of Biomedicine and Research Program in Genome-Scale Biology, Department of Pediatrics, PO Box 22 (Stenbäckinkatu 11), 00014 Helsinki, Finland
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Children's Hospital, University of Helsinki, Program for Women's Health, Department of Obstetrics and Gynecology, Department of Pathology, Institute of Biomedicine and Research Program in Genome-Scale Biology, Department of Pediatrics, PO Box 22 (Stenbäckinkatu 11), 00014 Helsinki, Finland
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Children's Hospital, University of Helsinki, Program for Women's Health, Department of Obstetrics and Gynecology, Department of Pathology, Institute of Biomedicine and Research Program in Genome-Scale Biology, Department of Pediatrics, PO Box 22 (Stenbäckinkatu 11), 00014 Helsinki, Finland
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Children's Hospital, University of Helsinki, Program for Women's Health, Department of Obstetrics and Gynecology, Department of Pathology, Institute of Biomedicine and Research Program in Genome-Scale Biology, Department of Pediatrics, PO Box 22 (Stenbäckinkatu 11), 00014 Helsinki, Finland
Children's Hospital, University of Helsinki, Program for Women's Health, Department of Obstetrics and Gynecology, Department of Pathology, Institute of Biomedicine and Research Program in Genome-Scale Biology, Department of Pediatrics, PO Box 22 (Stenbäckinkatu 11), 00014 Helsinki, Finland
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regulates cardiac BCL2 gene expression in vitro and in vivo . FASEB Journal 20 800 – 802 . Kobayashi S Volden P Timm D Mao K Xu X Liang Q 2009 Transcription factor GATA4 inhibits doxorubicin-induced autophagy and cardiomyocyte death
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. Cancer Research 56 1545 – 1550 . Heath-Engel HM Chang NC Shore GC 2008 The endoplasmic reticulum in apoptosis and autophagy: role of the BCL-2 protein family . Oncogene 27 6419 – 6433 . Hetz CA 2007 ER stress signaling and the BCL
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maintaining cellular energy supply required an AMPK-dependent and p53-dependent switch to autophagy in vivo and glycolysis in vitro . In the absence of functional p53, this switch is impaired and leads to an energy deficit and growth inhibition. In the
Barts and the London School of Medicine, Department of Endocrinology and Internal Medicine, Division of Endocrinology and Metabolism, Internal Medicine, Institute of Endocrinology and Metabolism, Centre for Endocrinology, London, UK
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Barts and the London School of Medicine, Department of Endocrinology and Internal Medicine, Division of Endocrinology and Metabolism, Internal Medicine, Institute of Endocrinology and Metabolism, Centre for Endocrinology, London, UK
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Barts and the London School of Medicine, Department of Endocrinology and Internal Medicine, Division of Endocrinology and Metabolism, Internal Medicine, Institute of Endocrinology and Metabolism, Centre for Endocrinology, London, UK
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R Aggarwal BB Kondo Y 2007 Evidence that curcumin suppresses the growth of malignant gliomas in vitro and in vivo through induction of autophagy: role of Akt and extracellular signal-regulated kinase signaling pathways . Molecular
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Department of Pediatric Hematology and Oncology, Service Central de Biophysique et de Médecine Nucléaire, Département d'Oncologie Moléculaire, Program in Reproductive and Adult Endocrinology, 2nd Medical School, Charles University and University Hospital Motol, Prague, Czech Republic
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Medicine 360 1361 – 1362 . (author reply 1362) ( doi:10.1056/NEJMc090088 ). Eng CH Abraham RT 2010 Glutaminolysis yields a metabolic by-product that stimulates autophagy . Autophagy 6 968 – 970 . ( doi:10.4161/auto.6.7.13082 ). Eng C
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Department of Cellular and Molecular, Faculty of Biological Sciences, PUC, Santiago, Chile
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Millennium Institute on Immunology and Immunotherapy, PUC, Santiago, Chile
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Millennium Institute on Immunology and Immunotherapy, PUC, Santiago, Chile
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Millennium Institute on Immunology and Immunotherapy, PUC, Santiago, Chile
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Millennium Institute on Immunology and Immunotherapy, PUC, Santiago, Chile
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mevalonate and cholesterol synthesis and blocking protein prenylation by downstream depletion of FPP and GGP ( Berndt et al . 2011 ). Treating mut-TP53 HGS-ovC cell lines with increasing concentrations of statins induces autophagy, cell death and drug