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D Grahame Hardie
D Grahame Hardie
Division of Cell Signalling & Immunology, School of Life Sciences, University of Dundee, Dundee, Scotland, UK
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different if he had been using cultured tumour cells! Another interesting feature of Fig. 4 is the high flux from malate to pyruvate via malic enzyme (Mal → Pyr; 10 nmol/min/mg). Along with the oxidative branch of the pentose phosphate pathway (G6P → P5P
Francesca Ruggieri
Francesca Ruggieri
Division of Oncology, Medical University of Graz, Graz, Austria
Research Unit “Non-Coding RNAs and Genome Editing in Cancer”, Division of Oncology, Medical University of Graz, Graz, Austria
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Research Unit “Non-Coding RNAs and Genome Editing in Cancer”, Division of Oncology, Medical University of Graz, Graz, Austria
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Katharina Jonas
Katharina Jonas
Division of Oncology, Medical University of Graz, Graz, Austria
Research Unit “Non-Coding RNAs and Genome Editing in Cancer”, Division of Oncology, Medical University of Graz, Graz, Austria
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Research Unit “Non-Coding RNAs and Genome Editing in Cancer”, Division of Oncology, Medical University of Graz, Graz, Austria
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Manuela Ferracin
Manuela Ferracin
Department of Experimental, Diagnostic and Specialty Medicine (DIMES), University of Bologna, Bologna, Italy
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Michael Dengler
Michael Dengler
Division of Oncology, Medical University of Graz, Graz, Austria
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Vanessa Jӓger
Vanessa Jӓger
Division of Oncology, Medical University of Graz, Graz, Austria
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Martin Pichler
Martin Pichler
Division of Oncology, Medical University of Graz, Graz, Austria
Research Unit “Non-Coding RNAs and Genome Editing in Cancer”, Division of Oncology, Medical University of Graz, Graz, Austria
Department of Hematology and Oncology, Medical Faculty, University of Augsburg, Augsburg, Germany
Translational Oncology, University Hospital of Augsburg, Augsburg, Germany
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Research Unit “Non-Coding RNAs and Genome Editing in Cancer”, Division of Oncology, Medical University of Graz, Graz, Austria
Department of Hematology and Oncology, Medical Faculty, University of Augsburg, Augsburg, Germany
Translational Oncology, University Hospital of Augsburg, Augsburg, Germany
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-34a ( Xiao et al. 2016 ), miR-30d-5p ( He et al. 2018 ), miR-449a ( Li et al. 2018 ), and miR-323a-3p ( Chen et al. 2018 ) as negative regulators of LDHA. All these miRNAs function as tumor suppressors in several cancers and their
