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Department of Endocrinology, Amsterdam University Medical Centers, VU University Medical Center, Amsterdam, The Netherlands
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vs aggressive insulinomas The low incidence of aggressive insulinomas has resulted in few data on genetic alterations in these tumors. The first evidence of biological differences between indolent and aggressive insulinomas was discovered by
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objective is then to relate the different β-cell changes in infants with CHI to the genetic alterations. Finally, the spectrum of β-cell changes that is associated with idiopathic or gastric bypass ANHH will be discussed. Table 1 Clinico
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differentiate insulinomatosis from MEN1-related insulinomas on the basis of conventional or functional imaging. Family history, additional endocrine disorders (mainly primary hyperparathyroidism and pituitary tumors) and genetic testing can be helpful to
ENETS Center of Excellence, University Hospital Tübingen, Tübingen, Germany
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finding suggested that GCHN, as we called the disease ( Sipos et al. 2015 ), seems to be caused by two different pathomechanisms, of which one is associated with a GCGR gene mutation, while the other is not and lacks so far any known genetic alteration
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). Alternatively, the term ‘persistent hyperinsulinemic hypoglycemia in infancy’ is used ( Glaser et al. 1989 , Sempoux & Kloppel 2023 ). This condition represents a group of clinically, genetically and histologically heterogeneous disorders, characterized by
