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signaling pathways in thyroid cancer. Cell apoptosis assay revealed that anlotinib induces apoptosis of thyroid cancer cells, partly through activating the TP53 pathway. Anlotinib also inhibits the migration of thyroid cancer cells, through interfering F
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Molecular Oncology Group, IMDEA Food Institute, CEI UAM-CSIC, Madrid, Spain
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Centro de Investigación Biomédica en Red de Cáncer (CIBERONC), Instituto de Salud Carlos III (ISCIII), Madrid, Spain
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thyroid development is required for thyrocyte precursor migration ( De Felice et al. 1998 , De Felice & Di Lauro 2004 , Parlato et al. 2004 , Fernandez et al. 2015 ). In the differentiated thyroid, Foxe1 is a transcriptional activator of the
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coworkers (2009) . In this study, we aimed to clarify the influence of NCOA1 in PCa metastasis formation. Using an RNAi approach to generate stable NCOA1 knockdown cell lines, we investigated the effect on migration and invasion as well as on
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and migration of PC cells and decreases cell adhesion Based on the higher incidence of PC relapse in patients with increased preoperative levels of sIL-6R and IL-6, we considered a possible involvement of IL-6TS in processes relevant to tumour
Department of Oncology, Zhoushan Hospital, Zhoushan, China
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TOP1MT may provide clues for therapeutic strategies by regulating the Warburg effect in tumors. Therefore, in this study, we sought to determine the role of TOP1MT in invasion and migration and elucidate the regulatory metabolic mechanisms through
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Australian Prostate Cancer Research Centre – Queensland, Department of Urologic Sciences, Institute of Health and Biomedical Innovation, Queensland University of Technology, Princess Alexandra Hospital, Translational Research Institute, Brisbane, Australia
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Australian Prostate Cancer Research Centre – Queensland, Department of Urologic Sciences, Institute of Health and Biomedical Innovation, Queensland University of Technology, Princess Alexandra Hospital, Translational Research Institute, Brisbane, Australia
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YKL40 in promoting the migration and invasion of PCa cells in addition to tumourigenicity. Materials and methods Cell culture All cell lines were maintained in phenol red-free RPMI-1640 media supplemented with 5% foetal bovine serum (FBS; Life
Division of Environmental Genetics and Molecular Toxicology, Division of Epidemiology and Biostatistics, Center for Environmental Genetics, Cancer Center, Department of Pathology, Department of Pathology and Laboratory Medicine, Department of Environmental Health
Division of Environmental Genetics and Molecular Toxicology, Division of Epidemiology and Biostatistics, Center for Environmental Genetics, Cancer Center, Department of Pathology, Department of Pathology and Laboratory Medicine, Department of Environmental Health
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Division of Environmental Genetics and Molecular Toxicology, Division of Epidemiology and Biostatistics, Center for Environmental Genetics, Cancer Center, Department of Pathology, Department of Pathology and Laboratory Medicine, Department of Environmental Health
Division of Environmental Genetics and Molecular Toxicology, Division of Epidemiology and Biostatistics, Center for Environmental Genetics, Cancer Center, Department of Pathology, Department of Pathology and Laboratory Medicine, Department of Environmental Health
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in PCa cells increased cell migration and invasiveness without affecting cell growth, while the expression of ERβ2 augmented only cell invasion. In aggregate, our data suggest that ERβ isoforms have variable functions and prognostic values in PCa
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Division of Surgery, Istituto Auxologico Italiano, Istituto di Ricovero e Cura a Carattere Scientifico (IRCCS), Milan, Italy
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-induced proliferation and migration of lymphatic endothelial cells and the formation of new lymphatic capillaries in which VEGFs are reported to play the most important role ( Joukov et al. 1996 ). Of note, several lines of evidence have shown that VEGFC plays a
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was performed to access the ability of migration and invasion. Further details of Transwell assay are presented in the Supplementary Materials and methods. Tumorigenicity assay in vivo Animal care and experiments were approved by the
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Department of Biomedical, Experimental and Clinical Sciences, University of Florence, Florence, Italy
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Molecular Endocrinology Laboratory, Department of Cellular and Molecular Medicine, KU Leuven, Leuven, Belgium
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decreased cell migration in androgen-dependent and CRPC cells ( Santer et al. 2011 ). Based on those previous results, this study aimed to investigate whether p300 is a possible new target in the context of chemotherapy resistance. Therefore, we analyzed