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Olson Center for Women's Health, Department of Obstetrics and Gynecology, The Fred and Pamela Buffett Cancer Center, Department of Pathology, Department of Ophthalmology and Visual Science, Department of Pathology, Omaha Veterans Affairs Medical Center, College of Animal Science and Technology
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Olson Center for Women's Health, Department of Obstetrics and Gynecology, The Fred and Pamela Buffett Cancer Center, Department of Pathology, Department of Ophthalmology and Visual Science, Department of Pathology, Omaha Veterans Affairs Medical Center, College of Animal Science and Technology
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Olson Center for Women's Health, Department of Obstetrics and Gynecology, The Fred and Pamela Buffett Cancer Center, Department of Pathology, Department of Ophthalmology and Visual Science, Department of Pathology, Omaha Veterans Affairs Medical Center, College of Animal Science and Technology
Olson Center for Women's Health, Department of Obstetrics and Gynecology, The Fred and Pamela Buffett Cancer Center, Department of Pathology, Department of Ophthalmology and Visual Science, Department of Pathology, Omaha Veterans Affairs Medical Center, College of Animal Science and Technology
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Olson Center for Women's Health, Department of Obstetrics and Gynecology, The Fred and Pamela Buffett Cancer Center, Department of Pathology, Department of Ophthalmology and Visual Science, Department of Pathology, Omaha Veterans Affairs Medical Center, College of Animal Science and Technology
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Olson Center for Women's Health, Department of Obstetrics and Gynecology, The Fred and Pamela Buffett Cancer Center, Department of Pathology, Department of Ophthalmology and Visual Science, Department of Pathology, Omaha Veterans Affairs Medical Center, College of Animal Science and Technology
Olson Center for Women's Health, Department of Obstetrics and Gynecology, The Fred and Pamela Buffett Cancer Center, Department of Pathology, Department of Ophthalmology and Visual Science, Department of Pathology, Omaha Veterans Affairs Medical Center, College of Animal Science and Technology
Olson Center for Women's Health, Department of Obstetrics and Gynecology, The Fred and Pamela Buffett Cancer Center, Department of Pathology, Department of Ophthalmology and Visual Science, Department of Pathology, Omaha Veterans Affairs Medical Center, College of Animal Science and Technology
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Olson Center for Women's Health, Department of Obstetrics and Gynecology, The Fred and Pamela Buffett Cancer Center, Department of Pathology, Department of Ophthalmology and Visual Science, Department of Pathology, Omaha Veterans Affairs Medical Center, College of Animal Science and Technology
Olson Center for Women's Health, Department of Obstetrics and Gynecology, The Fred and Pamela Buffett Cancer Center, Department of Pathology, Department of Ophthalmology and Visual Science, Department of Pathology, Omaha Veterans Affairs Medical Center, College of Animal Science and Technology
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heterozygosity, and short hairpin RNA (shRNA) knockdown of YAP increased migration and invasiveness, and enhanced tumor growth ( Yuan et al . 2008 ). Therefore, the expression and function of YAP in specific cancers requires further investigation. Granulosa cell
Department of Community Medicine, Division of Colon and Rectal Surgery, School of Medicine, Department of Physiology, Graduate Institute of Neural and Cognitive Sciences, Department of Internal Medicine, Department of Chinese Medicine, Comprehensive Breast Cancer Center, Department of Cell and Tissue Engineering, Preventive Medicine Center
Department of Community Medicine, Division of Colon and Rectal Surgery, School of Medicine, Department of Physiology, Graduate Institute of Neural and Cognitive Sciences, Department of Internal Medicine, Department of Chinese Medicine, Comprehensive Breast Cancer Center, Department of Cell and Tissue Engineering, Preventive Medicine Center
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Department of Community Medicine, Division of Colon and Rectal Surgery, School of Medicine, Department of Physiology, Graduate Institute of Neural and Cognitive Sciences, Department of Internal Medicine, Department of Chinese Medicine, Comprehensive Breast Cancer Center, Department of Cell and Tissue Engineering, Preventive Medicine Center
Department of Community Medicine, Division of Colon and Rectal Surgery, School of Medicine, Department of Physiology, Graduate Institute of Neural and Cognitive Sciences, Department of Internal Medicine, Department of Chinese Medicine, Comprehensive Breast Cancer Center, Department of Cell and Tissue Engineering, Preventive Medicine Center
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prognostic marker in colon cancer. In the present study, we investigated the intracellular signaling pathways involved in the BDNF-regulated cell motility in colon cancer cells. Our results indicated that BDNF induced cell migration by upregulating VEGF
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signaling pathways in thyroid cancer. Cell apoptosis assay revealed that anlotinib induces apoptosis of thyroid cancer cells, partly through activating the TP53 pathway. Anlotinib also inhibits the migration of thyroid cancer cells, through interfering F
Department of Pathology & Molecular Medicine, Queen’s University, Kingston, Ontario, Canada
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Department of Pathology & Molecular Medicine, Queen’s University, Kingston, Ontario, Canada
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Department of Pathology & Molecular Medicine, Queen’s University, Kingston, Ontario, Canada
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Department of Pathology & Molecular Medicine, Queen’s University, Kingston, Ontario, Canada
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Department of Medical Genetics, University of British Columbia, British Columbia Cancer Research Centre, Vancouver, British Columbia, Canada
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Department of Pathology & Molecular Medicine, Queen’s University, Kingston, Ontario, Canada
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, RET is not essential for cell survival but promotes changes in cell morphology and transcriptional pattern, and contributes to cell migration and invasion, consistent with a change to a more mesenchymal growth pattern. Interestingly, our data show that
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cancer cells, leptin increases proliferation ( Frankenberry et al . 2004 , 2006 , Housa et al . 2006 ). Leptin not only promotes the proliferation, but also the invasion and migration of hepatocellular and endometrial carcinoma cells ( Sharma
Istituto di Endocrinologia e Oncologia Sperimentale, Dipartimento di Biologia e Patologia Cellulare e Molecolare, Dipartimento di Scienze Farmaceutiche, Consiglio Nazionale delle Ricerche, Napoli, Italy
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Marzo 2002 , Portella et al . 2003 ). Moreover, it has been observed that cannabinoid receptors activation is able to inhibit vascular endothelial cell migration and to downregulate the expression and the activity of matrix metalloproteinase-2, a
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2013 , Klemm & Joyce 2014 ). Single cell and collective migration are hallmarks of cancer invasion and collective migration differs from single cell migration in that the cells remain connected as they move ( Frield & Gilmour 2009 , Wang et al
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Department of Biosciences, The Minerva Foundation Institute for Medical Research, Institute of Biomedicine/Physiology, Åbo Akademi University, Biocity, Artillerigatan 6, 20520 Turku, Finland
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Department of Biosciences, The Minerva Foundation Institute for Medical Research, Institute of Biomedicine/Physiology, Åbo Akademi University, Biocity, Artillerigatan 6, 20520 Turku, Finland
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al . 2011 ). Currently, there is no effective treatment available ( Smallridge et al . 2009 ). Sphingosine 1-phosphate (S1P) is a bioactive lipid regulating many cellular processes, including cell migration and proliferation. S1P may enhance or
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Molecular Oncology Group, IMDEA Food Institute, CEI UAM-CSIC, Madrid, Spain
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Centro de Investigación Biomédica en Red de Cáncer (CIBERONC), Instituto de Salud Carlos III (ISCIII), Madrid, Spain
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thyroid development is required for thyrocyte precursor migration ( De Felice et al. 1998 , De Felice & Di Lauro 2004 , Parlato et al. 2004 , Fernandez et al. 2015 ). In the differentiated thyroid, Foxe1 is a transcriptional activator of the
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coworkers (2009) . In this study, we aimed to clarify the influence of NCOA1 in PCa metastasis formation. Using an RNAi approach to generate stable NCOA1 knockdown cell lines, we investigated the effect on migration and invasion as well as on