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Esther Korpershoek, Bart-Jeroen Petri, Francien H van Nederveen, Winand N M Dinjens, Albert A Verhofstad, Wouter W de Herder, Sonja Schmid, Aurel Perren, Paul Komminoth, and Ronald R de Krijger

paragangliomas (sPGL; Lenders et al. 2005 ). Pheochromocytoma-associated syndromes include multiple endocrine neoplasia type 2 (MEN2), von Hippel–Lindau disease (VHL), neurofibromatosis-1, and the familial pheochromocytoma–paraganglioma (PCC–PGL) syndrome

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E Rapizzi, T Ercolino, L Canu, V Giaché, M Francalanci, C Pratesi, A Valeri, and M Mannelli

Introduction Pheochromocytoma (PHEO)/paraganglioma (PGL) are neural crest-derived tumors. According to their differentiation, they can be classified into two types: PGLs that are sympathetic in origin, catecholamine secreting, and mostly located in

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Annika Blank, Anja M Schmitt, Esther Korpershoek, Francien van Nederveen, Thomas Rudolph, Nicole Weber, Räto Thomas Strebel, Ronald de Krijger, Paul Komminoth, and Aurel Perren

paragangliomas (PGLs). About 30% of these tumours occur in familial tumour syndromes ( Neumann et al . 2002 , Amar et al . 2005 , Tischler 2008 , Komminoth 2009 ) including neurofibromatosis type 1 (NF1), von Hippel–Lindau disease (VHL), multiple endocrine

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Janos Geli, Nimrod Kiss, Fredrik Lanner, Theodoros Foukakis, Natalia Natalishvili, Olle Larsson, Per Kogner, Anders Höög, Geoffrey J Clark, Tomas J Ekström, Martin Bäckdahl, Filip Farnebo, and Catharina Larsson

related kinase (MST1) ( Khokhlatchev et al. 2002 , Praskova et al. 2004 ), suggesting that NORE1A and RASSF1A are involved in the same pro-apoptotic pathway. Pheochromocytomas and abdominal paragangliomas (the latter are here referred to as

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J Crona, F Beuschlein, K Pacak, and B Skogseid

pheochromocytomas (PCCs) and paragangliomas (PGLs, collectively denoted PPGLs), 177 on adrenocortical tumors and 18 that fell into a general adrenal tumor category. In this review, we have referenced 110 of these manuscripts and selected three prominent topics that

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N B Kiss, J Geli, F Lundberg, C Avci, D Velazquez-Fernandez, J Hashemi, G Weber, A Höög, T J Ekström, M Bäckdahl, and C Larsson

Introduction Abdominal tumors of the peripheral sympathetic nervous system include pheochromocytomas of the adrenal medulla and extra-adrenal paragangliomas originating from similar chromaffin cells in abdominal sympathetic paraganglia outside the

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Patricia L M Dahia, Roderick Clifton-Bligh, Anne-Paule Gimenez-Roqueplo, Mercedes Robledo, and Camilo Jimenez

priority areas for future research and consensus points were collated. The next sections summarize presentations and workshop discussions on metastatic pheochromocytomas and paragangliomas (mPPGLs) and conclude with proposed plans for the coming years and

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Nimrod B Kiss, Andreas Muth, Adam Andreasson, C Christofer Juhlin, Janos Geli, Martin Bäckdahl, Anders Höög, Bo Wängberg, Ola Nilsson, Håkan Ahlman, and Catharina Larsson

Introduction Pheochromocytomas are catecholamine-secreting tumors of the chromaffin cells of the adrenal medulla. Extra-adrenal abdominal paragangliomas (here referred to as abdominal paraganglioma or paraganglioma) are related to neuroendocrine

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P N Span, J U Rao, S B J Oude Ophuis, J W M Lenders, F C G J Sweep, P Wesseling, B Kusters, F H van Nederveen, R R de Krijger, A R M M Hermus, and H J L M Timmers

Introduction Paragangliomas (PGLs) are catecholamine-producing tumors that originate from chromaffin cells of the adrenal medulla (pheochromocytoma proper) or from sympathetic neuronal tissue in extra-adrenal locations of the abdomen or chest

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Alexandru Saveanu, Mihaela Muresan, Catherine De Micco, David Taieb, Anne-Laure Germanetti, Frederic Sebag, Jean-François Henry, Laurent Brunaud, Alain Enjalbert, Georges Weryha, and Anne Barlier

Introduction Pheochromocytomas (PCC) and paragangliomas (PGL) are neuroendocrine tumors derived from adrenal chromaffin cells and extra-adrenal paraganglia respectively ( Eisenhofer et al . 2008 ). These tumors cause variable secondary hypertension