K Brouwers FM Walther MM Linehan WM Munson PJ Mannelli M Goldstein DS Elkahloun AG 2004b Distinct gene expression profiles in norepinephrine- and epinephrine-producing hereditary and sporadic pheochromocytomas: activation of hypoxia
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Jens Waldmann, Volker Fendrich, Julia Holler, Malte Buchholz, Ernst Heinmöller, Peter Langer, Annette Ramaswamy, Birgit Samans, Martin K Walz, Matthias Rothmund, Detlef K Bartsch, and Emily P Slater
C Scheuba, K Kaserer, A Moritz, R Drosten, H Vierhapper, C Bieglmayer, O A Haas, and B Niederle
had to be at least twice the basal calcitonin levels ( Barbot et al . 1994 ). Table 1 Demographics – medullary thyroid cancer and C-cell hyperplasia C-cell hyperplasia (only) Medullary thyroid cancer Sporadic Hereditary Σ Sporadic Hereditary Σ N 122
Nicole Bechmann, Mats Leif Moskopp, Martin Ullrich, Bruna Calsina, Pål William Wallace, Susan Richter, Markus Friedemann, Katharina Langton, Stephanie M J Fliedner, Henri J L M Timmers, Svenja Nölting, Felix Beuschlein, Martin Fassnacht, Aleksander Prejbisz, Karel Pacak, Hans K Ghayee, Stefan R Bornstein, Peter Dieterich, Jens Pietzsch, Ben Wielockx, Mercedes Robledo, Nan Qin, and Graeme Eisenhofer
-0231 ) Eisenhofer G Huynh TT Pacak K Brouwers FM Walther MM Linehan WM Munson PJ Mannelli M Goldstein DS Elkahloun AG 2004 Distinct gene expression profiles in norepinephrine-and epinephrine-producing hereditary and sporadic pheochromocytomas
Yulong Li and William F Simonds
bronchial NET (sometimes referred to as ‘carcinoid tumors’); and 3) MEN2A-associated medullary thyroid cancer and pheochromocytoma. Because of space limitations, we favor the citation of recent review articles, wherein the curious reader can easily find
Zi Ying Tan, Taosheng Huang, and Joanne Ngeow
cell carcinoma, pheochromocytoma SDHA SDHB SDHD Hereditary paraganglioma–pheochromocytoma AD Paraganglioma–pheochromocytoma, gastrointestinal stromal tumour β-Catenin/WNT signalling pathway APC Familial adenomatous polyposis
Roland Därr, Joan Nambuba, Jaydira Del Rivero, Ingo Janssen, Maria Merino, Milena Todorovic, Bela Balint, Ivana Jochmanova, Josef T Prchal, Ronald M Lechan, Arthur S Tischler, Vera Popovic, Dragana Miljic, Karen T Adams, F Ryan Prall, Alexander Ling, Meredith R Golomb, Michael Ferguson, Naris Nilubol, Clara C Chen, Emily Chew, David Taïeb, Constantine A Stratakis, Tito Fojo, Chunzhang Yang, Electron Kebebew, Zhengping Zhuang, and Karel Pacak
Introduction Pheochromocytomas (PHEOs) and paragangliomas (PGLs) are rare catecholamine-producing neuroendocrine tumors (NETs) arising in or outside the adrenal medulla, respectively ( Lenders et al . 2005 ). By definition, a PHEO is an intra
Arthur Varoquaux, Yann le Fur, Alessio Imperiale, Antony Reyre, Marion Montava, Nicolas Fakhry, Izzie-Jacques Namer, Guy Moulin, Karel Pacak, Maxime Guye, and David Taïeb
succinate, which acts as an oncometabolite ( Selak et al . 2005 ). We, and others, have recently shown that ex vivo metabolomics studies are very reliable methods for classifying various pheochromocytomas (PHEOs)/PGLs according to their genetic background
M Montani, A M Schmitt, S Schmid, T Locher, P Saremaslani, P U Heitz, P Komminoth, and A Perren
. 2001 ) have recently been described in familial paragangliomas and pheochromocytomas. Several reasons make these genes candidate tumor suppressors for MTC: (1) allelic loss of the short and long arm of chromosome 1, the localization of SDHB and SDHC
Jessica L Geiger, Simion I Chiosea, Sue M Challinor, Marina N Nikiforova, and Julie E Bauman
T proto-oncogene on chromosome 10 ( Mulligan & Ponder 1995 , Santoro et al . 1995 ) and is further subclassified into three syndromes based on clinical phenotype: MEN2A (medullary thyroid cancer (MTC), pheochromocytoma, primary parathyroid
Elizabeth G Grubbs, Ronald M Lechan, Beth Edeiken-Monroe, Gilbert J Cote, Chardria Trotter, Arthur S Tischler, and Robert F Gagel
physician he saw very quickly pieced together a history consistent with a recently identified syndrome, multiple endocrine neoplasia type 2A – the association of medullary thyroid carcinoma (MTC), hyperparathyroidism and pheochromocytoma ( Steiner et al
