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Myriem Boufraqech Endocrine Oncology Branch Laboratory of Pathology National Cancer Institute, National Institutes of Health, Center for Cancer Research, Bethesda, Maryland 20892, USA

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Lisa Zhang Endocrine Oncology Branch Laboratory of Pathology National Cancer Institute, National Institutes of Health, Center for Cancer Research, Bethesda, Maryland 20892, USA

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Meenu Jain Endocrine Oncology Branch Laboratory of Pathology National Cancer Institute, National Institutes of Health, Center for Cancer Research, Bethesda, Maryland 20892, USA

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Dhaval Patel Endocrine Oncology Branch Laboratory of Pathology National Cancer Institute, National Institutes of Health, Center for Cancer Research, Bethesda, Maryland 20892, USA

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Ryan Ellis Endocrine Oncology Branch Laboratory of Pathology National Cancer Institute, National Institutes of Health, Center for Cancer Research, Bethesda, Maryland 20892, USA

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Yin Xiong Endocrine Oncology Branch Laboratory of Pathology National Cancer Institute, National Institutes of Health, Center for Cancer Research, Bethesda, Maryland 20892, USA

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Mei He Endocrine Oncology Branch Laboratory of Pathology National Cancer Institute, National Institutes of Health, Center for Cancer Research, Bethesda, Maryland 20892, USA

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Naris Nilubol Endocrine Oncology Branch Laboratory of Pathology National Cancer Institute, National Institutes of Health, Center for Cancer Research, Bethesda, Maryland 20892, USA

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Maria J Merino Endocrine Oncology Branch Laboratory of Pathology National Cancer Institute, National Institutes of Health, Center for Cancer Research, Bethesda, Maryland 20892, USA

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Electron Kebebew Endocrine Oncology Branch Laboratory of Pathology National Cancer Institute, National Institutes of Health, Center for Cancer Research, Bethesda, Maryland 20892, USA

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3′-UTR Reporter gene plasmid, pLightSwitch-Akt3-3′-UTR, was co-transfected with miR-C mimic or miR-145 mimic ( miRVana, Applied Biosystems) into cells using Lipofectamine 2000 Reagent (Life Technologies). Luciferase activity was measured 24 h after

Article Type:
Research Article
DOI:
https://doi.org/10.1530/ERC-14-0077
Volume/Issue:
Volume 21: Issue 4
Free access
Kathrin Zitzmann Department of Internal Medicine II, Institute of Molecular Animal Breeding and Biotechnology, University‐Hospital Munich-Grosshadern, Munich, Germany

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George Vlotides Department of Internal Medicine II, Institute of Molecular Animal Breeding and Biotechnology, University‐Hospital Munich-Grosshadern, Munich, Germany

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Stephan Brand Department of Internal Medicine II, Institute of Molecular Animal Breeding and Biotechnology, University‐Hospital Munich-Grosshadern, Munich, Germany

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Harald Lahm Department of Internal Medicine II, Institute of Molecular Animal Breeding and Biotechnology, University‐Hospital Munich-Grosshadern, Munich, Germany

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Gerald Spöttl Department of Internal Medicine II, Institute of Molecular Animal Breeding and Biotechnology, University‐Hospital Munich-Grosshadern, Munich, Germany

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Burkhard Göke Department of Internal Medicine II, Institute of Molecular Animal Breeding and Biotechnology, University‐Hospital Munich-Grosshadern, Munich, Germany

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Christoph J Auernhammer Department of Internal Medicine II, Institute of Molecular Animal Breeding and Biotechnology, University‐Hospital Munich-Grosshadern, Munich, Germany

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varies between different organs, with high expression levels in insulin-responsive tissues such as muscle and liver. In contrast, Akt3 expression is more restricted to neuronal tissue ( Stambolic & Woodgett 2006 , Mendoza & Blenis 2007 ). Although

Article Type:
Research Article
DOI:
https://doi.org/10.1530/ERC-12-0074
Volume/Issue:
Volume 19: Issue 3
Free access
Zhe Wang Department of Gastroenterology, Tongji Hospital, Tongji University School of Medicine, Shanghai, China
Division of Gastroenterology, Department of Medicine, Sidney Kimmel Comprehensive Cancer Center, Johns Hopkins University School of Medicine, Baltimore, Maryland, USA

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Ke Ma Division of Gastroenterology, Department of Medicine, Sidney Kimmel Comprehensive Cancer Center, Johns Hopkins University School of Medicine, Baltimore, Maryland, USA

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Steffie Pitts Cellular and Molecular Medicine Graduate Program, Johns Hopkins University School of Medicine, Baltimore, Maryland, USA

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Yulan Cheng Division of Gastroenterology, Department of Medicine, Sidney Kimmel Comprehensive Cancer Center, Johns Hopkins University School of Medicine, Baltimore, Maryland, USA

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Xi Liu Department of Pathology, The First Affiliated Hospital of Xi’ an Jiaotong University, Xi’ an, Shaanxi, China

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Xiquan Ke Department of Gastroenterology, The First Affiliated Hospital of Bengbu Medical College, Bengbu, Anhui, China

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Samuel Kovaka Department of Computer Science, Whiting School of Engineering, Johns Hopkins University, Baltimore, Maryland, USA

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Hassan Ashktorab Cancer Center, Howard University School of Medicine, Washington, District of Columbia, USA

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Duane T Smoot Department of Medicine, Meharry Medical College, Nashville, Tennessee, USA

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Michael Schatz Department of Computer Science, Whiting School of Engineering, Johns Hopkins University, Baltimore, Maryland, USA

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Zhirong Wang Department of Gastroenterology, Tongji Hospital, Tongji University School of Medicine, Shanghai, China

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Stephen J Meltzer Division of Gastroenterology, Department of Medicine, Sidney Kimmel Comprehensive Cancer Center, Johns Hopkins University School of Medicine, Baltimore, Maryland, USA

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functional assays using interference and overexpression of circNF1 revealed that circNF1 promotes cell proliferation. Finally, circNF1 functioned as a miR-16 sponge, derepressing the miR-16 target genes MAP7 and AKT3 . We conclude that circNF1

Article Type:
Research Article
DOI:
https://doi.org/10.1530/ERC-18-0478
Volume/Issue:
Volume 26: Issue 3
Free access
Lena Lapkina-Gendler Department of Human Molecular Genetics and Biochemistry, Sackler School of Medicine, Tel Aviv University, Tel Aviv, Israel

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Itai Rotem Department of Human Molecular Genetics and Biochemistry, Sackler School of Medicine, Tel Aviv University, Tel Aviv, Israel

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Metsada Pasmanik-Chor Bioinformatics Unit, George Wise Faculty of Life Sciences, Tel Aviv University, Tel Aviv, Israel

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David Gurwitz Department of Human Molecular Genetics and Biochemistry, Sackler School of Medicine, Tel Aviv University, Tel Aviv, Israel
Yoran Institute for Human Genome Research, Tel Aviv University, Tel Aviv, Israel

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Rive Sarfstein Department of Human Molecular Genetics and Biochemistry, Sackler School of Medicine, Tel Aviv University, Tel Aviv, Israel

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Zvi Laron Endocrine and Diabetes Research Unit, Schneider Children’s Medical Center, Petah Tikva, Israel

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Haim Werner Department of Human Molecular Genetics and Biochemistry, Sackler School of Medicine, Tel Aviv University, Tel Aviv, Israel
Yoran Institute for Human Genome Research, Tel Aviv University, Tel Aviv, Israel

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, versican, and AKT3 mRNA levels were measured by RQ-PCR, using appropriate primers ( Table 1 ). For control purpose, actin mRNA levels were measured. Table 1 Sequences of primers employed in RQ-PCR analyses. Primer Sequence Annealing

Article Type:
Research Article
DOI:
https://doi.org/10.1530/ERC-16-0054
Volume/Issue:
Volume 23: Issue 5
Free access
Vincenzo Condello Department of Surgical, Medical, Molecular Pathology and Critical Area, University Hospital of Pisa, Pisa, Italy
Department of Pathology, University of Pittsburgh, Pittsburgh, Pennsylvania, USA

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Filomena Cetani Endocrine Unit, University Hospital of Pisa, Pisa, Italy

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Maria Denaro Department of Surgical, Medical, Molecular Pathology and Critical Area, University Hospital of Pisa, Pisa, Italy

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Liborio Torregrossa Division of Surgical Pathology, University Hospital of Pisa, Pisa, Italy

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Elena Pardi Department of Clinical and Experimental Medicine, University of Pisa, Pisa, Italy

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Paolo Piaggi Department of Information Engineering, University of Pisa, Pisa, Italy

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Simona Borsari Department of Clinical and Experimental Medicine, University of Pisa, Pisa, Italy

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Anello Marcello Poma Department of Surgical, Medical, Molecular Pathology and Critical Area, University Hospital of Pisa, Pisa, Italy

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Lucia Anna Muscarella Fondazione IRCCS Casa Sollievo della Sofferenza Hospital, Laboratory of Oncology, San Giovanni Rotondo (FG), Italy

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Paolo Graziano Fondazione IRCCS Casa Sollievo della Sofferenza Hospital, Unit of Pathology, San Giovanni Rotondo (FG), Italy

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Maria Grazia Chiofalo Fondazione IRCCS G. Pascale, Thyroid and Parathyroid Surgery Unit, Istituto Nazionale Tumori, Naples, Italy

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Andrea Repaci Endocrinology Unit, Sant’Orsola-Malpighi Hospital, University of Bologna, Bologna, Italy

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Giovanni Tallini Department of Medicine, Molecular Diagnostic Unit, Azienda USL di Bologna, University of Bologna School of Medicine, Bologna, Italy

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Francesco Boi Department of Medical Sciences and Public Health, Endocrinology Unit, University of Cagliari, Cagliari, Italy

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Gabriele Materazzi Department of Surgical, Medical, Molecular Pathology and Critical Area, University Hospital of Pisa, Pisa, Italy

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Fulvio Basolo Department of Surgical, Medical, Molecular Pathology and Critical Area, University Hospital of Pisa, Pisa, Italy

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Claudio Marcocci Department of Clinical and Experimental Medicine, University of Pisa, Pisa, Italy

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,VEGFC C. Metastatic PCs vs Non-metastatic PCs  HOTAIR regulatory pathway Cancer, cellular growth, proliferation and development 9.88 1.3E−10 0.26 20/156 (13%) 0/156 (0%) 31/156 (20%) 105/156 (67%) AKT3,CDH1,ERBB2,MMP12,MMP14,MMP24

Article Type:
Research Article
DOI:
https://doi.org/10.1530/ERC-20-0450
Volume/Issue:
Volume 28: Issue 2
Restricted access
Susan Ha Departments of, Pharmacology, Urology and NYU Cancer Institute, Psychiatry, Department of Nephrology, New York University School of Medicine, 550 First Avenue, MSB424, New York, New York 10016, USA
Departments of, Pharmacology, Urology and NYU Cancer Institute, Psychiatry, Department of Nephrology, New York University School of Medicine, 550 First Avenue, MSB424, New York, New York 10016, USA

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Rachel Ruoff Departments of, Pharmacology, Urology and NYU Cancer Institute, Psychiatry, Department of Nephrology, New York University School of Medicine, 550 First Avenue, MSB424, New York, New York 10016, USA
Departments of, Pharmacology, Urology and NYU Cancer Institute, Psychiatry, Department of Nephrology, New York University School of Medicine, 550 First Avenue, MSB424, New York, New York 10016, USA

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Nicole Kahoud Departments of, Pharmacology, Urology and NYU Cancer Institute, Psychiatry, Department of Nephrology, New York University School of Medicine, 550 First Avenue, MSB424, New York, New York 10016, USA
Departments of, Pharmacology, Urology and NYU Cancer Institute, Psychiatry, Department of Nephrology, New York University School of Medicine, 550 First Avenue, MSB424, New York, New York 10016, USA

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Thomas F Franke Departments of, Pharmacology, Urology and NYU Cancer Institute, Psychiatry, Department of Nephrology, New York University School of Medicine, 550 First Avenue, MSB424, New York, New York 10016, USA
Departments of, Pharmacology, Urology and NYU Cancer Institute, Psychiatry, Department of Nephrology, New York University School of Medicine, 550 First Avenue, MSB424, New York, New York 10016, USA

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Susan K Logan Departments of, Pharmacology, Urology and NYU Cancer Institute, Psychiatry, Department of Nephrology, New York University School of Medicine, 550 First Avenue, MSB424, New York, New York 10016, USA
Departments of, Pharmacology, Urology and NYU Cancer Institute, Psychiatry, Department of Nephrology, New York University School of Medicine, 550 First Avenue, MSB424, New York, New York 10016, USA

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AR levels under the experimental conditions. Alternatively, Akt i , which is preferential for Akt isoforms 1 and 2 ( DeFeo-Jones et al . 2005 , Lindsley et al . 2005 ), may not inhibit all of the Akt3 isoform that is present in VCaP cells. VCaP

Article Type:
Research Article
DOI:
https://doi.org/10.1530/ERC-10-0204
Volume/Issue:
Volume 18: Issue 2
Free access
Mona Alharbi Exosome Biology Laboratory, Centre for Clinical Diagnostics, University of Queensland Centre for Clinical Research, Royal Brisbane and Women’s Hospital, The University of Queensland, Brisbane Queensland, Australia

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Felipe Zuñiga Department of Clinical Biochemistry and Immunology, Faculty of Pharmacy, University of Concepción, Concepción, Chile

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Omar Elfeky Exosome Biology Laboratory, Centre for Clinical Diagnostics, University of Queensland Centre for Clinical Research, Royal Brisbane and Women’s Hospital, The University of Queensland, Brisbane Queensland, Australia

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Dominic Guanzon Exosome Biology Laboratory, Centre for Clinical Diagnostics, University of Queensland Centre for Clinical Research, Royal Brisbane and Women’s Hospital, The University of Queensland, Brisbane Queensland, Australia

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Andrew Lai Exosome Biology Laboratory, Centre for Clinical Diagnostics, University of Queensland Centre for Clinical Research, Royal Brisbane and Women’s Hospital, The University of Queensland, Brisbane Queensland, Australia

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Gregory E Rice Exosome Biology Laboratory, Centre for Clinical Diagnostics, University of Queensland Centre for Clinical Research, Royal Brisbane and Women’s Hospital, The University of Queensland, Brisbane Queensland, Australia
Maternal-Fetal Medicine, Department of Obstetrics and Gynecology, Ochsner Clinic Foundation, New Orleans, Louisiana, USA
Perinatology Research Branch, NICHD/NIH, Wayne State University, Detroit, Michigan, USA

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Lewis Perrin Mater Research Institute, University of Queensland, Translational Research Institute, Woolloongabba, Queensland, Australia
Mater Ovarian Cancer Research Collaborative, Mater Adult Hospital, South Brisbane, Queensland, Australia

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John Hooper Mater Research Institute, University of Queensland, Translational Research Institute, Woolloongabba, Queensland, Australia
Mater Ovarian Cancer Research Collaborative, Mater Adult Hospital, South Brisbane, Queensland, Australia

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Carlos Salomon Exosome Biology Laboratory, Centre for Clinical Diagnostics, University of Queensland Centre for Clinical Research, Royal Brisbane and Women’s Hospital, The University of Queensland, Brisbane Queensland, Australia
Department of Clinical Biochemistry and Immunology, Faculty of Pharmacy, University of Concepción, Concepción, Chile
Maternal-Fetal Medicine, Department of Obstetrics and Gynecology, Ochsner Clinic Foundation, New Orleans, Louisiana, USA

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levels and MDR1 mRNA and PTEN cysteine protease serine protease Li et al. (2015)  miR-489 SKOV3/CDDP and OVCAR3/CDDP ↓ AKT3 Annexin calmodulin non-receptor serine/threonine protein kinase transfer/carrier protein Wu et al. (2014

Article Type:
Review Article
DOI:
https://doi.org/10.1530/ERC-18-0019
Volume/Issue:
Volume 25: Issue 12
Free access
M Bullock Hormones and Cancer Group, Cancer Genetics Laboratory, Kolling Institute of Medical Research, Royal North Shore Hospital, Pacific Highway Saint Leonards, Sydney, New South Wales 2065, Australia

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specific nature of the most common mutations described: (1) HER2 receptor amplification; (2) PI3CA (p110) activating mutations; (3) deletion of INPP4B gene; (4) PTEN inactivating mutations/deletions; and (5) AKT1/AKT3 activating mutations. FOXO gene family

Article Type:
Review Article
DOI:
https://doi.org/10.1530/ERC-15-0461
Volume/Issue:
Volume 23: Issue 2
Free access
Zvi Laron Endocrinology and Diabetes Research Unit, Schneider Children’s Medical Center, Petah Tikva, Israel

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Haim Werner Department of Human Molecular Genetics and Biochemistry, Sackler School of Medicine, Tel Aviv University, Tel Aviv, Israel

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matrix disassembly Genes downregulated in LS Biological role Cyclin A1 Cell cycle regulation AKT3 Apoptosis Transcription factor Sp1 Gene regulation Olfactory receptor Detection of odor molecules Nephronectin

Article Type:
Review Article
Collections:
Growth Hormone in Endocrine Cancers
DOI:
https://doi.org/10.1530/ERC-22-0394
Volume/Issue:
Volume 30: Issue 9
Free access
Belinda J Petri Department of Biochemistry & Molecular Genetics, University of Louisville School of Medicine Louisville, Kentucky, USA

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Kellianne M Piell Department of Biochemistry & Molecular Genetics, University of Louisville School of Medicine Louisville, Kentucky, USA

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Ali E Wilt Department of Biochemistry & Molecular Genetics, University of Louisville School of Medicine Louisville, Kentucky, USA

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Alexa D Howser Department of Biochemistry & Molecular Genetics, University of Louisville School of Medicine Louisville, Kentucky, USA

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Laura Winkler Department of Biochemistry & Molecular Genetics, University of Louisville School of Medicine Louisville, Kentucky, USA

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Mattie R Whitworth Department of Biochemistry & Molecular Genetics, University of Louisville School of Medicine Louisville, Kentucky, USA

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Bailey L Valdes Department of Biochemistry & Molecular Genetics, University of Louisville School of Medicine Louisville, Kentucky, USA

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Norman L Lehman Department of Biochemistry & Molecular Genetics, University of Louisville School of Medicine Louisville, Kentucky, USA
Pathology and Laboratory Medicine, University of Louisville, Louisville, Kentucky, USA
The Brown Cancer Center, University of Louisville, Louisville, Kentucky, USA

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Brian F Clem Department of Biochemistry & Molecular Genetics, University of Louisville School of Medicine Louisville, Kentucky, USA
The Brown Cancer Center, University of Louisville, Louisville, Kentucky, USA

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Carolyn M Klinge Department of Biochemistry & Molecular Genetics, University of Louisville School of Medicine Louisville, Kentucky, USA
The Brown Cancer Center, University of Louisville, Louisville, Kentucky, USA
Center for Integrative Environmental Health Sciences (CIEHS), University of Louisville, Louisville, Kentucky, USA

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protein. Taken together with previous reports, our new data suggest a common inverse relationship between miR-145-5p and PSAT1 in some cancers. Although miR-145-5p was negatively correlated with CCAT2 and AKT3 which have higher expression in TAM

Article Type:
Research Article
DOI:
https://doi.org/10.1530/ERC-23-0148
Volume/Issue:
Volume 30: Issue 11
Restricted access