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FAM dye-labelled (vascular endothelial growth factor A ( VEGFA ), Hs000900055_m1; α -SMA , Hs00426835_g1; PTH , Hs00757710_1g and CCND1 , Hs00765553_m1, Applied Biosystems, Life Technologies), 10 μl of 2× TaqMan Gene Expression MasterMix (Applied
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. Further expanding tumours show cellular hypoxia which leads to a rise in HIF-1 protein expression leading to the production of angiogenic factors, such as vascular endothelial growth factor-A (VEGF-A), which is involved in the sprouting of new vessels into
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exposure. We confirmed ZEB 1/2 , validated VEGFA, FBLN5 , and TIMP2 as direct targets of miR-200c in MSMC, LSMC, and SKLM-S1, and showed that overexpression of miR-200c caused phenotypic modification of MSMC and LSMC, but not SKLM-S1, and inhibited
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stimuli for angiogenesis has yielded numerous candidates including vascular endothelial growth factor A (VEGF-A), fibroblast growth factor-2 (FGF-2), transforming growth factor α (TGF-α), TGF-β, hepatocyte growth factor, tumour necrosis factor α
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Department of Biology, Turku Graduate School of Biomedical Sciences, Laboratory of Animal Physiology, Hospital for Children and Adolescents, Minerva Foundation Institute for Medical Research, Åbo Akademi University, Tykistökatu 6A, 20520 Turku, Finland
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Department of Biology, Turku Graduate School of Biomedical Sciences, Laboratory of Animal Physiology, Hospital for Children and Adolescents, Minerva Foundation Institute for Medical Research, Åbo Akademi University, Tykistökatu 6A, 20520 Turku, Finland
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amounts of both VEGF-A and -C compared with FRO cells, but S1P stimulates VEGF-A secretion in both cell lines. We show that VEGFR-2 signalling is involved in S1P-induced Akt phosphorylation and migration of ML-1 cells. Furthermore, VEGFR-2 regulates S1P
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Endocrine Surgery, IRCCS Ospedale San Raffaele, Milan, Italy
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Department of Biomedical, Surgical and Dental Sciences, University of Milan, Milan, Italy
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Department of Biomedical, Surgical and Dental Sciences, University of Milan, Milan, Italy
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Division of Pathology, Fondazione IRCCS Ca’ Granda Ospedale Maggiore Policlinico, Milan, Italy
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Endocrinology and Diabetology Service, IRCCS Istituto Ortopedico Galeazzi, Milan, Italy
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expressed at the surface of endothelial cells. One well-known activator of angiogenesis is the secreted protein vascular endothelial growth factor A (VEGFA) ( Ferrara 2009 ). We previously demonstrated that the tumor-associated myofibroblasts (TAMs) are
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Endocrinology Department, “C.I. Parhon” National Institute of Endocrinology, Bucharest, Romania
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Faculté de Médecine Lyon Est, Université Lyon 1, Lyon, France
Department of Endocrinology, Reference center for rare pituitary disease (HYPO), Groupement Hospitalier EST, Hospices Civils de Lyon, University of Lyon, Lyon, France
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’-AGGACATAGGGGAGCAGTTTCAG-3’), Ki67 (Fw 5’-GCTGTCCTCAAGACAATCATCA-3’; Rev 5’-GGCGTTATCCCAGGAGACT-3’), Gdf9 (Fw 5’-CAACCAGGTGACAGGACCG-3’; Rev 5’-AGCAAGTGTTCCATGGCAGT-3’), Vegfa (Fw 5’-CTGCTCTCTTGGGTGCACTG-3’; Rev 5’-GCAGCCTGGGACCACTTG-3’), Vegfb (Fw 5
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Genomics Research Centre, Institute of Health and Biomedical Innovation, Faculty of Health, Queensland University of Technology, Brisbane, Queensland, Australia
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plays an important role in activating the proteins and pro-angiogenic factors involved ( Fig. 1 ). Vascular endothelial growth factor A ( VEGF-A ), above all other known pro-angiogenic factors, induces sprouting of new blood vessels. In thyroid
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, when OVX females were used, E2 treatment increased tumour-related blood vessel network, while no modulation was observed in males. VEGFC, VEGFD, VEGFA and bFGF, the main prolymphangiogenic and proangiogenic factors, were measured by Milliplex assay in
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′CAGAGGATGACCTGCCAACA; 3′GCCCTTCCTTTCCTGTGTCAT. SLUG primers: 5′TTTCTGGGCTGGCCAAACAT; 3′TTCTCCCCCGTGTGAGTTCTA. TWIST1 primers: 5′GCCGGAGACCTAGATGTCATT; 3′CCCACGCCCTGTTTCTTTGA. VEGFA primers: 5′AGGCCAGCACATAGGAGAGA; 3′TTTCTTGCGCTTTCGTTTTT. VEGFD primers: 5