Search Results
Universitat Internacional de Catalunya (UIC), Barcelona, Spain
Search for other papers by Elena Valassi in
Google Scholar
PubMed
Search for other papers by Frédéric Castinetti in
Google Scholar
PubMed
Search for other papers by Amandine Ferriere in
Google Scholar
PubMed
Search for other papers by Stylianos Tsagarakis in
Google Scholar
PubMed
Search for other papers by Richard A Feelders in
Google Scholar
PubMed
Search for other papers by Romana T Netea-Maier in
Google Scholar
PubMed
Search for other papers by Michael Droste in
Google Scholar
PubMed
Search for other papers by Christian J Strasburger in
Google Scholar
PubMed
Search for other papers by Dominique Maiter in
Google Scholar
PubMed
Search for other papers by Darko Kastelan in
Google Scholar
PubMed
Search for other papers by Philippe Chanson in
Google Scholar
PubMed
Universitat Autònoma de Barcelona (UAB), Barcelona, Spain
Search for other papers by Susan M Webb in
Google Scholar
PubMed
Search for other papers by Frank Demtröder in
Google Scholar
PubMed
Search for other papers by Valdis Pirags in
Google Scholar
PubMed
Search for other papers by Olivier Chabre in
Google Scholar
PubMed
Search for other papers by Holger Franz in
Google Scholar
PubMed
Universitat Autònoma de Barcelona (UAB), Barcelona, Spain
Search for other papers by Alicia Santos in
Google Scholar
PubMed
Search for other papers by Martin Reincke in
Google Scholar
PubMed
( Fountas & Karavitaki 2020 ). A recent consensus recognized the presence of an expanding adenoma on imaging as the most accurate diagnostic criterion of NS and, accordingly, proposed the definition of ‘corticotroph tumor progression after bilateral
Search for other papers by Anna Bagnato in
Google Scholar
PubMed
Search for other papers by Francesca Spinella in
Google Scholar
PubMed
Search for other papers by Laura Rosanò in
Google Scholar
PubMed
patients in advanced stages is still associated by low survival rates, the development of new treatment protocols depends on improved knowledge of the molecular mechanisms controlling tumor progression ( Naora & Montell 2005 ). Cancer invasion is a state
Search for other papers by Yvonne Fierz in
Google Scholar
PubMed
Search for other papers by Ruslan Novosyadlyy in
Google Scholar
PubMed
Search for other papers by Archana Vijayakumar in
Google Scholar
PubMed
Search for other papers by Shoshana Yakar in
Google Scholar
PubMed
Search for other papers by Derek LeRoith in
Google Scholar
PubMed
the setting of type 2 diabetes are largely unknown. To study the effect of mTOR blockade on type 2 diabetes-induced mammary tumor progression, we employed a hyperinsulinemic mouse model of type 2 diabetes, the female MKR mice. These mice overexpress
Search for other papers by A Bardin in
Google Scholar
PubMed
Search for other papers by N Boulle in
Google Scholar
PubMed
Search for other papers by G Lazennec in
Google Scholar
PubMed
Search for other papers by F Vignon in
Google Scholar
PubMed
Search for other papers by P Pujol in
Google Scholar
PubMed
feature of estrogen-dependent tumor progression in clinical studies Analysis of ERα and ERβ expression in estrogen-sensitive cancers (Tables 1–4) In breast tissues, several studies have indicated an increase in ERα/ERβ mRNA
Search for other papers by Annu Makker in
Google Scholar
PubMed
Search for other papers by Madhu Mati Goel in
Google Scholar
PubMed
in CTNNB1 , PPP21A , and p53 genes have been suggested to contribute to tumor progression from EEC to UEC ( Kuhn et al . 2014 ). Although the overall 5-year survival rate in patients with stage I EEC approaches 90%, ∼30% of these tumors are high
Search for other papers by Zane Hammoud in
Google Scholar
PubMed
Search for other papers by Bailin Tan in
Google Scholar
PubMed
Search for other papers by Sunil Badve in
Google Scholar
PubMed
Search for other papers by Robert M Bigsby in
Google Scholar
PubMed
that estrogen increased tumor grade in males ( P <0.001), but not in ovariectomized females ( Fig. 4 ). Figure 4 Effect of estradiol on tumor progression. Using a scale described by Jackson et al . (2005) for grading mouse lung tumors, histological
Search for other papers by Louis de Mestier in
Google Scholar
PubMed
Search for other papers by Clarisse Dromain in
Google Scholar
PubMed
Search for other papers by Gaspard d'Assignies in
Google Scholar
PubMed
Search for other papers by Jean-Yves Scoazec in
Google Scholar
PubMed
Search for other papers by Nathalie Lassau in
Google Scholar
PubMed
Search for other papers by Rachida Lebtahi in
Google Scholar
PubMed
Search for other papers by Hedia Brixi in
Google Scholar
PubMed
Search for other papers by Emmanuel Mitry in
Google Scholar
PubMed
Search for other papers by Rosine Guimbaud in
Google Scholar
PubMed
Search for other papers by Frédéric Courbon in
Google Scholar
PubMed
Search for other papers by Michèle d'Herbomez in
Google Scholar
PubMed
Search for other papers by Guillaume Cadiot in
Google Scholar
PubMed
SD: no criteria for CR, PR, or PD and no symptomatic deterioration attributed to tumor progression PD: ≥10% increase in the sum of diameters, increase in size of existing intra-tumoral nodules, new lesion, or intra-tumor nodule Chun
Search for other papers by Teresa Ramone in
Google Scholar
PubMed
Search for other papers by Cristina Romei in
Google Scholar
PubMed
Search for other papers by Raffaele Ciampi in
Google Scholar
PubMed
Search for other papers by Roberta Casalini in
Google Scholar
PubMed
Search for other papers by Angelo Valetto in
Google Scholar
PubMed
Search for other papers by Veronica Bertini in
Google Scholar
PubMed
Search for other papers by Francesco Raimondi in
Google Scholar
PubMed
Search for other papers by Anthony Onoja in
Google Scholar
PubMed
Search for other papers by Alessandro Prete in
Google Scholar
PubMed
Search for other papers by Antonio Matrone in
Google Scholar
PubMed
Search for other papers by Carla Gambale in
Google Scholar
PubMed
Search for other papers by Paolo Piaggi in
Google Scholar
PubMed
Search for other papers by Liborio Torregrossa in
Google Scholar
PubMed
Search for other papers by Clara Ugolini in
Google Scholar
PubMed
Search for other papers by Rossella Elisei in
Google Scholar
PubMed
et al. 2007 ). In fact, the amplification of chromosomal regions containing oncogenes involved in tumor progression or the loss of regions containing TSG could justify the correlation between SCNA and metastatic progression and poor outcomes of the
Search for other papers by Nikki A Ford in
Google Scholar
PubMed
Search for other papers by Nomeli P Nunez in
Google Scholar
PubMed
Search for other papers by Valerie B Holcomb in
Google Scholar
PubMed
Department of Nutritional Sciences, Department of Molecular Carcinogenesis, Dell Pediatric Research Institute, University of Texas, 1400 Barbara Jordan Boulevard, DPRI 2.834, Austin, Texas 78722, USA
Search for other papers by Stephen D Hursting in
Google Scholar
PubMed
). Furthermore, the Met1 cell line, derived from a spontaneous tumor from a polyoma middle-T antigen transgenic mouse, progressively loses ER and progesterone receptor positivity with tumor progression, therefore providing a rapid and relevant model for studying
Department of Cancer Biology, Department of Urology, Kimmel Cancer Center, Thomas Jefferson University, 233 S 10th Street, BLSB 1008, Philadelphia, Pennsylvania 19107, USA
Search for other papers by Clay E S Comstock in
Google Scholar
PubMed
Department of Cancer Biology, Department of Urology, Kimmel Cancer Center, Thomas Jefferson University, 233 S 10th Street, BLSB 1008, Philadelphia, Pennsylvania 19107, USA
Department of Cancer Biology, Department of Urology, Kimmel Cancer Center, Thomas Jefferson University, 233 S 10th Street, BLSB 1008, Philadelphia, Pennsylvania 19107, USA
Department of Cancer Biology, Department of Urology, Kimmel Cancer Center, Thomas Jefferson University, 233 S 10th Street, BLSB 1008, Philadelphia, Pennsylvania 19107, USA
Search for other papers by Karen E Knudsen in
Google Scholar
PubMed
with the conclusion that tumor progression is largely driven by resurgent AR activity, hormone therapy ‘failure’ is typically first detected by a rise in PSA despite the maintenance of ligand depletion and AR-directed therapeutic regimens, indicating